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Molecular Cancer Research : MCR|July 9, 2009
Elevated poly-(ADP-ribose)-polymerase activity sensitizes retinoblastoma-deficient cells to DNA damage-induced necrosisHuiping Liu, James R Knabb, Benjamin T Spike, et al.Nature Reviews. Cancer|January 11, 2013
MDM2, MDMX and p53 in oncogenesis and cancer therapyMark Wade, Yao-Cheng Li, Geoffrey M WahlCancer Prevention Research (Philadelphia, Pa.)|July 1, 2015
A Critical Examination of the "Bad Luck" Explanation of Cancer RiskAndrii I Rozhok, Geoffrey M Wahl, James DeGregoriTrends in Cell Biology|February 23, 2010
The p53 orchestra: Mdm2 and Mdmx set the toneMark Wade, Yunyuan V Wang, Geoffrey M WahlBreast Cancer Research and Treatment|January 11, 2015
Luminal progenitor and fetal mammary stem cell expression features predict breast tumor response to neoadjuvant chemotherapyAdam D Pfefferle, Benjamin T Spike, Geoff M Wahl, et al.Journal of Virology|October 15, 2003
Metaphase chromosome tethering is necessary for the DNA synthesis and maintenance of oriP plasmids but is insufficient for transcription activation by Epstein-Barr nuclear antigen 1John Sears, John Kolman, Geoffrey M Wahl, et al.Plos One|December 7, 2017
Reprogramming pancreatic stellate cells via p53 activation: A putative target for pancreatic cancer therapyMaya Saison-Ridinger, Kathleen E DelGiorno, Tejia Zhang, et al.Cell Cycle (Georgetown, Tex.)|July 8, 2008
BH3 activation blocks Hdmx suppression of apoptosis and cooperates with Nutlin to induce cell deathMark Wade, Luo Wei Rodewald, Joaquín M Espinosa, et al.Proceedings of the National Academy of Sciences of the United States of America|July 12, 2005
The C-terminal lysines fine-tune P53 stress responses in a mouse model but are not required for stability control or transactivationKurt A Krummel, Crystal J Lee, Franck Toledo, et al.Nucleic Acids Research|July 28, 2006
RMCE-ASAP: a gene targeting method for ES and somatic cells to accelerate phenotype analysesFranck Toledo, Chung-Wen Liu, Crystal J Lee, et al.Pageof 11