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Methods in Enzymology|January 27, 2011
The emerging role of histone deacetylases (HDACs) in UPR regulationSoumen Kahali, Bhaswati Sarcar, Prakash ChinnaiyanJournal of Neuro-Oncology|February 6, 2010
Vorinostat enhances the cytotoxic effects of the topoisomerase I inhibitor SN38 in glioblastoma cell linesBhaswati Sarcar, Soumen Kahali, Prakash ChinnaiyanJournal of Cancer Treatment & Diagnosis|August 14, 2019
Hypoxia-Induced Autophagy Degrades Stromal Lumican into Tumor Microenvironment of Pancreatic Ductal Adenocarcinoma: A Mini-ReviewBhaswati Sarcar, Xinqun Li, Jason B FlemingAdvances in Experimental Medicine and Biology|June 27, 2020
Stellate Cells in the Tumor MicroenvironmentDavid Roife, Bhaswati Sarcar, Jason B FlemingPlos One|January 4, 2013
Targeting the unfolded protein response in glioblastoma cells with the fusion protein EGF-SubAAntony Prabhu, Bhaswati Sarcar, Soumen Kahali, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 6, 2012
Class I histone deacetylases localize to the endoplasmic reticulum and modulate the unfolded protein responseSoumen Kahali, Bhaswati Sarcar, Antony Prabhu, et al.Virology|April 6, 2004
Hepatitis C virus NS5A mediated STAT3 activation requires co-operation of Jak1 kinaseBhaswati Sarcar, Asish K Ghosh, Robert Steele, et al.Molecular Cancer Therapeutics|October 14, 2011
Targeting radiation-induced G(2) checkpoint activation with the Wee-1 inhibitor MK-1775 in glioblastoma cell linesBhaswati Sarcar, Soumen Kahali, Antony H Prabhu, et al.Neuro-Oncology|February 26, 2015
Ras-mediated modulation of pyruvate dehydrogenase activity regulates mitochondrial reserve capacity and contributes to glioblastoma tumorigenesisAntony Prabhu, Bhaswati Sarcar, C Ryan Miller, et al.Neoplasia (New York, N.Y.)|January 15, 2010
Activation of the unfolded protein response contributes toward the antitumor activity of vorinostatSoumen Kahali, Bhaswati Sarcar, Bin Fang, et al.Pageof 3