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Cellular and Molecular Life Sciences : CMLS|July 6, 2007
Tryptophan degradation in autoimmune diseasesC A Opitz, W Wick, L Steinman, et al.Der Nervenarzt|July 18, 2012
[Treatment of brain tumor patients: hyperthermia, hyperbaric oxygenation, electric fields or nanoparticles]M Platten, W WickJournal of Neuro-Oncology|November 22, 2001
Glioma cell invasion: regulation of metalloproteinase activity by TGF-betaW Wick, M Platten, M WellerDer Nervenarzt|September 3, 2021
[Brain tumor immunotherapy-Possibilities and challenges of personalization]M Platten, L Bunse, W WickMicroscopy Research and Technique|February 15, 2001
Malignant glioma biology: role for TGF-beta in growth, motility, angiogenesis, and immune escapeM Platten, W Wick, M WellerBritish Journal of Pharmacology|November 13, 2001
N-[3,4-dimethoxycinnamoyl]-anthranilic acid (tranilast) suppresses microglial inducible nitric oxide synthase (iNOS) expression and activity induced by interferon-gamma (IFN-gamma)M Platten, W Wick, J Wischhusen, et al.International Journal of Cancer|June 8, 2001
N-[3,4-dimethoxycinnamoyl]-anthranilic acid (tranilast) inhibits transforming growth factor-beta relesase and reduces migration and invasiveness of human malignant glioma cellsM Platten, C Wild-Bode, W Wick, et al.Biochemical and Biophysical Research Communications|February 19, 2000
Transforming growth factors beta(1) (TGF-beta(1)) and TGF-beta(2) promote glioma cell migration via Up-regulation of alpha(V)beta(3) integrin expressionM Platten, W Wick, C Wild-Bode, et al.Pageof 31