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Cell Death and Differentiation|October 1, 1996
Bile acid activation of the gadd153 promoter and of p53-independent apoptosis: relevance to colon cancerZ Y Zheng, H Bernstein, C Bernstein, et al.Toxicology Letters|September 21, 2000
Increased susceptibility of cells to inducible apoptosis during growth from early to late log phase: an important caveat for in vitro apoptosis researchD Washo-Stultz, C Crowley, C M Payne, et al.Cell Biology and Toxicology|December 3, 2005
Mitochondrial perturbation attenuates bile acid-induced cytotoxicityC M Payne, C L Crowley-Weber, K Dvorak, et al.Diseases of the Esophagus : Official Journal of the International Society for Diseases of the Esophagus|September 21, 2006
Esophageal acid exposure at pH < or = 2 is more common in Barrett's esophagus patients and is associated with oxidative stressK Dvorak, R Fass, R Dekel, et al.Cell Death and Differentiation|April 14, 1999
The stress-response proteins poly(ADP-ribose) polymerase and NF-kappaB protect against bile salt-induced apoptosisC M Payne, C Crowley, D Washo-Stultz, et al.Carcinogenesis|August 5, 2006
Deoxycholate induces mitochondrial oxidative stress and activates NF-kappaB through multiple mechanisms in HCT-116 colon epithelial cellsC M Payne, C Weber, C Crowley-Skillicorn, et al.Journal of Virology|April 1, 1971
Reversion of frameshift mutations stimulated by lesions in early function genes of bacteriophage T4H BernsteinOecologia|March 18, 2017
Prey and predator emigration responses in the acarine system Tetranychus urticae-Phytoseiulus persimilisC BernsteinBiophysical Journal|December 1, 1970
A comparison of the number of nucleotides per unit length in Escherichia coli and phage T4 chromosomesC BernsteinPageof 137