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C Frank Bennett

Showing results (101-110 of 169) with videos related to

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Genomics|February 4, 2015
Rescue of gene-expression changes in an induced mouse model of spinal muscular atrophy by an antisense oligonucleotide that promotes inclusion of SMN2 exon 7John F Staropoli, Huo Li, Seung J Chun, et al.
EMBO Molecular Medicine|September 10, 2013
Pathological impact of SMN2 mis-splicing in adult SMA miceKentaro Sahashi, Karen K Y Ling, Yimin Hua, et al.
Nucleic Acids Research|August 22, 2013
Rational design of antisense oligonucleotides targeting single nucleotide polymorphisms for potent and allele selective suppression of mutant Huntingtin in the CNSMichael E Østergaard, Amber L Southwell, Holly Kordasiewicz, et al.
Biochemistry|October 30, 2010
Allele-selective inhibition of mutant huntingtin expression with antisense oligonucleotides targeting the expanded CAG repeatKeith T Gagnon, Hannah M Pendergraff, Glen F Deleavey, et al.
Neuron|April 19, 2014
Muscle expression of mutant androgen receptor accounts for systemic and motor neuron disease phenotypes in spinal and bulbar muscular atrophyConstanza J Cortes, Shuo-Chien Ling, Ling T Guo, et al.
Molecular Cell|August 28, 2010
The nuclear-retained noncoding RNA MALAT1 regulates alternative splicing by modulating SR splicing factor phosphorylationVidisha Tripathi, Jonathan D Ellis, Zhen Shen, et al.
Nature Communications|December 23, 2020
MaTAR25 lncRNA regulates the Tensin1 gene to impact breast cancer progressionKung-Chi Chang, Sarah D Diermeier, Allen T Yu, et al.
The Journal of Pharmacology and Experimental Therapeutics|September 3, 2015
Identification and characterization of modified antisense oligonucleotides targeting DMPK in mice and nonhuman primates for the treatment of myotonic dystrophy type 1Sanjay K Pandey, Thurman M Wheeler, Samantha L Justice, et al.
The Journal of Clinical Investigation|August 1, 2006
Antisense oligonucleotide therapy for neurodegenerative diseaseRichard A Smith, Timothy M Miller, Koji Yamanaka, et al.
Parkinsonism & Related Disorders|August 19, 2015
Chronic and acute LRRK2 silencing has no long-term behavioral effects, whereas wild-type and mutant LRRK2 overexpression induce motor and cognitive deficits and altered regulation of dopamine releaseMattia Volta, Stefano Cataldi, Dayne Beccano-Kelly, et al.
Pageof 17

Showing results (101-110 of 169) with videos related to

Sort By:
Pageof 17
Genomics|February 4, 2015
Rescue of gene-expression changes in an induced mouse model of spinal muscular atrophy by an antisense oligonucleotide that promotes inclusion of SMN2 exon 7John F Staropoli, Huo Li, Seung J Chun, et al.
EMBO Molecular Medicine|September 10, 2013
Pathological impact of SMN2 mis-splicing in adult SMA miceKentaro Sahashi, Karen K Y Ling, Yimin Hua, et al.
Nucleic Acids Research|August 22, 2013
Rational design of antisense oligonucleotides targeting single nucleotide polymorphisms for potent and allele selective suppression of mutant Huntingtin in the CNSMichael E Østergaard, Amber L Southwell, Holly Kordasiewicz, et al.
Biochemistry|October 30, 2010
Allele-selective inhibition of mutant huntingtin expression with antisense oligonucleotides targeting the expanded CAG repeatKeith T Gagnon, Hannah M Pendergraff, Glen F Deleavey, et al.
Neuron|April 19, 2014
Muscle expression of mutant androgen receptor accounts for systemic and motor neuron disease phenotypes in spinal and bulbar muscular atrophyConstanza J Cortes, Shuo-Chien Ling, Ling T Guo, et al.
Molecular Cell|August 28, 2010
The nuclear-retained noncoding RNA MALAT1 regulates alternative splicing by modulating SR splicing factor phosphorylationVidisha Tripathi, Jonathan D Ellis, Zhen Shen, et al.
Nature Communications|December 23, 2020
MaTAR25 lncRNA regulates the Tensin1 gene to impact breast cancer progressionKung-Chi Chang, Sarah D Diermeier, Allen T Yu, et al.
The Journal of Pharmacology and Experimental Therapeutics|September 3, 2015
Identification and characterization of modified antisense oligonucleotides targeting DMPK in mice and nonhuman primates for the treatment of myotonic dystrophy type 1Sanjay K Pandey, Thurman M Wheeler, Samantha L Justice, et al.
The Journal of Clinical Investigation|August 1, 2006
Antisense oligonucleotide therapy for neurodegenerative diseaseRichard A Smith, Timothy M Miller, Koji Yamanaka, et al.
Parkinsonism & Related Disorders|August 19, 2015
Chronic and acute LRRK2 silencing has no long-term behavioral effects, whereas wild-type and mutant LRRK2 overexpression induce motor and cognitive deficits and altered regulation of dopamine releaseMattia Volta, Stefano Cataldi, Dayne Beccano-Kelly, et al.
Pageof 17