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The Surgical Clinics of North America|June 1, 1994
Physiologic support of the septic patientR H Demling, C Lalonde, K Ikegami
The Journal of Burn Care & Rehabilitation|December 24, 1997
Plasma catalase and glutathione levels are decreased in response to inhalation injuryC LaLonde, U Nayak, J Hennigan, et al.
The Journal of Burn Care & Rehabilitation|September 1, 1996
Antioxidants prevent the cellular deficit produced in response to burn injuryC LaLonde, U Nayak, J Hennigan, et al.
Circulatory Shock|January 1, 1994
Lung and systemic oxidant and antioxidant activity after graded smoke exposure in the ratC Lalonde, L Picard, C Campbell, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|July 1, 1986
Lung dysfunction after thermal injury in relation to prostanoid and oxygen radical releaseL J Jin, C Lalonde, R H Demling
Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1988
Endotoxin causes hydrogen peroxide-induced lung lipid peroxidation and prostanoid productionR Demling, C Lalonde, A Seekamp, et al.
The Journal of Trauma|January 1, 1986
Effect of anesthesia and positive pressure ventilation on early postburn hemodynamic instabilityL J Jin, C Lalonde, R H Demling
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