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Biochemical and Biophysical Research Communications|March 2, 1999
Early decrease of IIx myosin heavy chain transcripts in Duchenne muscular dystrophyM Pedemonte, C Sandri, S Schiaffino, et al.International Journal of Oral and Maxillofacial Surgery|February 5, 2014
New TASER injuries: lacrimal canaliculus laceration and ethmoid bone fractureA de Runz, C Minetti, M Brix, et al.The Laryngoscope|May 1, 1985
T-lymphocytes phenotype and functions in patients with head and neck cancerI Pierri, G Cordone, S Rogna, et al.La Pediatria Medica E Chirurgica : Medical and Surgical Pediatrics|January 1, 1986
[Ranitidine in primary peptic ulcer in childhood complicated by acute hemorrhage]G Cordone, V Venzano, F Ricci, et al.Neurology|March 1, 1991
Abnormal tau-reactive filaments in olfactory mucosa in biopsy specimens of patients with probable Alzheimer's diseaseM Tabaton, S Cammarata, G L Mancardi, et al.The Journal of Biological Chemistry|August 28, 1999
Phenotypic behavior of caveolin-3 mutations that cause autosomal dominant limb girdle muscular dystrophy (LGMD-1C). Retention of LGMD-1C caveolin-3 mutants within the golgi complexF Galbiati, D Volonte, C Minetti, et al.The Journal of Biological Chemistry|September 7, 2000
Limb-girdle muscular dystrophy (LGMD-1C) mutants of caveolin-3 undergo ubiquitination and proteasomal degradation. Treatment with proteasomal inhibitors blocks the dominant negative effect of LGMD-1C mutanta and rescues wild-type caveolin-3F Galbiati, D Volonte, C Minetti, et al.Neurology|February 26, 2004
Caveolinopathies: mutations in caveolin-3 cause four distinct autosomal dominant muscle diseasesS E Woodman, F Sotgia, F Galbiati, et al.Acta Otorhinolaryngologica Italica : Organo Ufficiale Della Societa Italiana Di Otorinolaringologia E Chirurgia Cervico-Facciale|August 6, 1999
[Mid-latency auditory evoked responses in Alzheimer's disease: evaluation of P1 and P3 waves]A Cordone, M Bavazzano, A Sismondini, et al.Pageof 11