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Continuum (Minneapolis, Minn.)|December 4, 2019
Sporadic Inclusion Body Myositis and Other Rimmed Vacuolar MyopathiesConrad C WeihlCurrent Alzheimer Research|January 13, 2011
Valosin containing protein associated fronto-temporal lobar degeneration: clinical presentation, pathologic features and pathogenesisC C WeihlNeurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|February 15, 2013
Monitoring autophagy in the treatment of protein aggregate diseases: steps toward identifying autophagic biomarkersConrad C WeihlThe Journal of Clinical Investigation|November 23, 2011
Another VCP interactor: NF is enoughConrad C WeihlAutophagy|August 17, 2017
Regulation of SQSTM1/p62 via UBA domain ubiquitination and its role in diseaseYouJin Lee, Conrad C WeihlAutophagy|October 11, 2021
Loss-of-function mutation in VCP mimics the characteristic pathology as in FTLD-TARDBPAbubakar Wani, Conrad C WeihlMuscle & Nerve|August 25, 2006
Motor neuron disease associated with copper deficiencyConrad C Weihl, Glenn LopateCurrent Opinion in Neurology|June 9, 2025
Myofibrillar myopathy: towards a mechanism-based definition as a Z-disk-opathyMichio Inoue, Conrad C WeihlCurrent Opinion in Neurology|July 29, 2010
Sporadic inclusion body myositis: possible pathogenesis inferred from biomarkersConrad C Weihl, Alan PestronkJournal of Cell Science|August 23, 2014
The VCP/p97 system at a glance: connecting cellular function to disease pathogenesisHemmo Meyer, Conrad C WeihlPageof 14