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American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|October 28, 2011
Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanismsJingchuan Zhang, Robert C RitterBrain Research|July 12, 2005
Reduced CCK-induced Fos expression in the hindbrain, nodose ganglia, and enteric neurons of rats lacking CCK-1 receptorsMihai Covasa, Robert C RitterResearch Communications in Chemical Pathology and Pharmacology|December 1, 1981
Relative muscarinic synaptic activation by anionic, neutral or cationic liposomesC Ritter, R J RutmanThe American Journal of Physiology|October 1, 1988
Capsaicin attenuates suppression of sham feeding induced by intestinal nutrientsD P Yox, R C RitterThe American Journal of Physiology|September 1, 1986
Area postrema lesions: cause of overingestion is not altered visceral nerve functionG L Edwards, R C RitterPostgraduate Medicine|February 1, 1994
Peripartum complications. Hemorrhage, embolism, hypertension, and infectionD C Ritter, R D deShazoActa Biologica Et Medica Germanica|January 1, 1975
[Variation of mechanical and biochemical parameters of pressure-loaded rabbit hearts following chronic administration of dipyridamole]C Ritter, M Weiss, W FörsterPeptides|April 2, 2003
Cholecystokinin activates specific enteric neurons in the rat small intestineAyman I Sayegh, Robert C RitterBrain Research|December 8, 1980
Increased feeding and hyperglycemia elicited by intracerebroventricular 5-thioglucoseP G Slusser, R C RitterAppetite|September 1, 1983
Feeding elicited by 2-deoxyglucose occurs in the absence of reduced glucose oxidationV K Nonavinakere, R C RitterPageof 40