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Science'S STKE : Signal Transduction Knowledge Environment|May 4, 2006
Where do you think you are going? The NMDA-D1 receptor trapCarlos Cepeda, Michael S LevineNeuron|May 15, 2012
2B or not 2B: a tail of two NMDA receptor subunitsCarlos Cepeda, Michael S LevineThe Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry|November 17, 2020
Synaptic Dysfunction in Huntington's Disease: Lessons from Genetic Animal ModelsCarlos Cepeda, Michael S LevineFuture Neurology|April 21, 2015
Evidence from the R6/2 Mouse Model of Huntington's Disease for Using Abnormal Brain Metabolism as a Biomarker for Evaluating Therapeutic Approaches for TreatmentAnna Parievsky, Carlos Cepeda, Michael S LevineNeuron|February 16, 2010
Location, location, location: contrasting roles of synaptic and extrasynaptic NMDA receptors in Huntington's diseaseMichael S Levine, Carlos Cepeda, Véronique M AndréASN Neuro|June 30, 2010
Alterations in striatal synaptic transmission are consistent across genetic mouse models of Huntington's diseaseDamian M Cummings, Carlos Cepeda, Michael S LevineCNS Neuroscience & Therapeutics|April 22, 2010
Dopamine and glutamate in Huntington's disease: A balancing actVéronique M André, Carlos Cepeda, Michael S LevineJournal of Neurophysiology|May 25, 2007
Altered corticostriatal neurotransmission and modulation in dopamine transporter knock-down miceNanping Wu, Carlos Cepeda, Xiaoxi Zhuang, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 26, 2018
Striatal Direct and Indirect Pathway Output Structures Are Differentially Altered in Mouse Models of Huntington's DiseaseJoshua Barry, Garnik Akopian, Carlos Cepeda, et al.Frontiers in Neuroscience|November 21, 2022
Calcium imaging: A versatile tool to examine Huntington's disease mechanisms and progressionJoshua Barry, Allison Peng, Michael S Levine, et al.Pageof 20