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Nature Metabolism|July 29, 2020
Dihydroxyacetone phosphate signals glucose availability to mTORC1Jose M Orozco, Patrycja A Krawczyk, Sonia M Scaria, et al.Immunity|June 22, 2017
Germinal Center Selection and Affinity Maturation Require Dynamic Regulation of mTORC1 KinaseJonatan Ersching, Alejo Efeyan, Luka Mesin, et al.Aging Cell|July 26, 2014
Depletion of Rictor, an essential protein component of mTORC2, decreases male lifespanDudley W Lamming, Maria M Mihaylova, Pekka Katajisto, et al.Neurobiology of Disease|December 24, 2017
A mouse model of DEPDC5-related epilepsy: Neuronal loss of Depdc5 causes dysplastic and ectopic neurons, increased mTOR signaling, and seizure susceptibilityChristopher J Yuskaitis, Brandon M Jones, Rachel L Wolfson, et al.Cell Metabolism|May 7, 2020
A Nutrient-Sensing Transition at Birth Triggers Glucose-Responsive Insulin SecretionAharon Helman, Andrew L Cangelosi, Jeffrey C Davis, et al.Molecular Cell|April 30, 2003
GbetaL, a positive regulator of the rapamycin-sensitive pathway required for the nutrient-sensitive interaction between raptor and mTORDo-Hyung Kim, D D Sarbassov, Siraj M Ali, et al.ACS Chemical Biology|April 13, 2012
Selective ATP-competitive inhibitors of TOR suppress rapamycin-insensitive function of TORC2 in Saccharomyces cerevisiaeQingsong Liu, Tao Ren, Tara Fresques, et al.Cell|October 21, 2017
mTORC1 Activator SLC38A9 Is Required to Efflux Essential Amino Acids from Lysosomes and Use Protein as a NutrientGregory A Wyant, Monther Abu-Remaileh, Rachel L Wolfson, et al.Proceedings of the National Academy of Sciences of the United States of America|January 23, 2021
Genome-wide CRISPR screens reveal multitiered mechanisms through which mTORC1 senses mitochondrial dysfunctionKendall J Condon, Jose M Orozco, Charles H Adelmann, et al.Nature|March 28, 2014
Metabolic determinants of cancer cell sensitivity to glucose limitation and biguanidesKıvanç Birsoy, Richard Possemato, Franziska K Lorbeer, et al.Pageof 26