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Chaitra Sathyaprakash

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Frontiers in Cell and Developmental Biology|September 19, 2025
Stem/progenitor cell-based therapy for Duchenne muscular dystrophyTsukasa Tominari, Chaitra Sathyaprakash, Yoshitsugu Aoki
Frontiers in Physiology|March 25, 2022
Editorial: Challenges and Opportunities for Neuromuscular Disease Modelling Using Urine-derived Stem CellsChaitra Sathyaprakash, Katsuhiko Kunitake, Yoshitsugu Aoki
Methods in Molecular Biology (Clifton, N.J.)|November 18, 2022
Quantitative Evaluation of Exon Skipping in Urine-Derived Cells for Duchenne Muscular DystrophyKatsuhiko Kunitake, Chaitra Sathyaprakash, Norio Motohashi, et al.
Molecular Pharmacology|October 24, 2014
Activation of human 5-hydroxytryptamine type 3 receptors via an allosteric transmembrane siteStuart J Lansdell, Chaitra Sathyaprakash, Anne Doward, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 28, 2025
Exon-Skipping Using Antisense Oligonucleotides for Laminin-Alpha2-Deficient Muscular DystrophyEri Takeuchi, Chaitra Sathyaprakash, Hotake Takizawa, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 28, 2025
In Vivo Evaluation of Single- and Multi-Exon-Skipping in mdx52 MiceChaitra Sathyaprakash, Eri Takeuchi, Hotake Takizawa, et al.
Methods in Molecular Biology (Clifton, N.J.)|September 1, 2020
Development of LNA Gapmer Oligonucleotide-Based Therapy for ALS/FTD Caused by the C9orf72 Repeat ExpansionChaitra Sathyaprakash, Raquel Manzano, Miguel A Varela, et al.
Stem Cell Reports|April 25, 2020
Impairment of Mitochondrial Calcium Buffering Links Mutations in C9ORF72 and TARDBP in iPS-Derived Motor Neurons from Patients with ALS/FTDRuxandra Dafinca, Paola Barbagallo, Lucy Farrimond, et al.
Nucleic Acids Research|January 23, 2025
Tissue-specific modulation of CRISPR activity by miRNA-sensing guide RNAsAntonio Garcia-Guerra, Chaitra Sathyaprakash, Olivier G de Jong, et al.
Brain and Neuroscience Advances|April 1, 2022
Reducing voltage-dependent potassium channel Kv3.4 levels ameliorates synapse loss in a mouse model of Alzheimer's diseaseJie Yeap, Chaitra Sathyaprakash, Jamie Toombs, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Frontiers in Cell and Developmental Biology|September 19, 2025
Stem/progenitor cell-based therapy for Duchenne muscular dystrophyTsukasa Tominari, Chaitra Sathyaprakash, Yoshitsugu Aoki
Frontiers in Physiology|March 25, 2022
Editorial: Challenges and Opportunities for Neuromuscular Disease Modelling Using Urine-derived Stem CellsChaitra Sathyaprakash, Katsuhiko Kunitake, Yoshitsugu Aoki
Methods in Molecular Biology (Clifton, N.J.)|November 18, 2022
Quantitative Evaluation of Exon Skipping in Urine-Derived Cells for Duchenne Muscular DystrophyKatsuhiko Kunitake, Chaitra Sathyaprakash, Norio Motohashi, et al.
Molecular Pharmacology|October 24, 2014
Activation of human 5-hydroxytryptamine type 3 receptors via an allosteric transmembrane siteStuart J Lansdell, Chaitra Sathyaprakash, Anne Doward, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 28, 2025
Exon-Skipping Using Antisense Oligonucleotides for Laminin-Alpha2-Deficient Muscular DystrophyEri Takeuchi, Chaitra Sathyaprakash, Hotake Takizawa, et al.
Methods in Molecular Biology (Clifton, N.J.)|July 28, 2025
In Vivo Evaluation of Single- and Multi-Exon-Skipping in mdx52 MiceChaitra Sathyaprakash, Eri Takeuchi, Hotake Takizawa, et al.
Methods in Molecular Biology (Clifton, N.J.)|September 1, 2020
Development of LNA Gapmer Oligonucleotide-Based Therapy for ALS/FTD Caused by the C9orf72 Repeat ExpansionChaitra Sathyaprakash, Raquel Manzano, Miguel A Varela, et al.
Stem Cell Reports|April 25, 2020
Impairment of Mitochondrial Calcium Buffering Links Mutations in C9ORF72 and TARDBP in iPS-Derived Motor Neurons from Patients with ALS/FTDRuxandra Dafinca, Paola Barbagallo, Lucy Farrimond, et al.
Nucleic Acids Research|January 23, 2025
Tissue-specific modulation of CRISPR activity by miRNA-sensing guide RNAsAntonio Garcia-Guerra, Chaitra Sathyaprakash, Olivier G de Jong, et al.
Brain and Neuroscience Advances|April 1, 2022
Reducing voltage-dependent potassium channel Kv3.4 levels ameliorates synapse loss in a mouse model of Alzheimer's diseaseJie Yeap, Chaitra Sathyaprakash, Jamie Toombs, et al.
Pageof 2