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Drug Discovery Today. Disease Models|July 25, 2008
The role of Na dysregulation in cardiac disease and how it impacts electrophysiologyBrian O'Rourke, Christoph MaackBasic Research in Cardiology|July 28, 2007
Excitation-contraction coupling and mitochondrial energeticsChristoph Maack, Brian O'RourkeCirculation Research|June 26, 2004
Partial inhibition of sodium/calcium exchange restores cellular calcium handling in canine heart failureIon A Hobai, Christoph Maack, Brian O'RourkeCirculation Research|November 20, 2004
Cardiac sodium-calcium exchanger is regulated by allosteric calcium and exchanger inhibitory peptide at distinct sitesChristoph Maack, Anand Ganesan, Agnieszka Sidor, et al.The Journal of Biological Chemistry|June 2, 2007
Sequential opening of mitochondrial ion channels as a function of glutathione redox thiol statusMiguel A Aon, Sonia Cortassa, Christoph Maack, et al.Circulation Research|January 7, 2006
Beta-adrenergic stimulation of L-type Ca2+ channels in cardiac myocytes requires the distal carboxyl terminus of alpha1C but not serine 1928Anand N Ganesan, Christoph Maack, David C Johns, et al.Biochemical and Biophysical Research Communications|March 2, 2005
Reverse engineering the L-type Ca2+ channel alpha1c subunit in adult cardiac myocytes using novel adenoviral vectorsAnand N Ganesan, Brian O'Rourke, Christoph Maack, et al.Circulation Research|June 17, 2006
Elevated cytosolic Na+ decreases mitochondrial Ca2+ uptake during excitation-contraction coupling and impairs energetic adaptation in cardiac myocytesChristoph Maack, Sonia Cortassa, Miguel A Aon, et al.Circulation|March 31, 2010
Elevated cytosolic Na+ increases mitochondrial formation of reactive oxygen species in failing cardiac myocytesMichael Kohlhaas, Ting Liu, Andreas Knopp, et al.Pageof 38