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Pharmacology & Therapeutics|April 26, 2011
Mitochondria are sources of metabolic sink and arrhythmiasFadi G Akar, Brian O'RourkeExpert Opinion on Investigational Drugs|June 4, 2004
The potential of Na+/Ca2+ exchange blockers in the treatment of cardiac diseaseIon A Hobai, Brian O'RourkeCardiovascular Research|July 13, 2010
Cardiac mitochondria and arrhythmiasDavid A Brown, Brian O'RourkeJournal of Molecular and Cellular Cardiology|January 24, 2009
Mitochondrial Ca2+ uptake: tortoise or hare?Brian O'Rourke, Lothar A BlatterMed (New York, N.Y.)|April 2, 2026
Emerging therapeutic directions for inotropic therapy in heart failureGianluigi Pironti, Christoph Maack, Daniel C Andersson, et al.Deutsche Medizinische Wochenschrift (1946)|February 18, 2016
[Cardiopulmonary Comorbidities]Frederik Seiler, Albrecht von Hardenberg, Michael Böhm, et al.European Heart Journal|July 5, 2015
Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implicationsThomas Münzel, Tommaso Gori, John F Keaney, et al.European Heart Journal|July 24, 2025
Mitochondrial cardiomyopathies: pathogenesis, diagnosis, and treatmentChristoph Maack, Jan Dudek, Edoardo Bertero, et al.Journal of Molecular and Cellular Cardiology|December 8, 2020
Mitochondrial Ca2+ in heart failure: Not enough or too much?Brian O'Rourke, Deepthi Ashok, Ting LiuInternational Journal of Technology Assessment in Health Care|May 14, 2020
The new definition of health technology assessment: A milestone in international collaborationBrian O'Rourke, Wija Oortwijn, Tara Schuller, et al.Pageof 38