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Clara Abraham

Showing results (31-40 of 87) with videos related to

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Journal of Immunology (Baltimore, Md. : 1950)|July 15, 2020
Disease Risk-Associated Genetic Variants in <i>STAT1</i> and <i>STAT4</i> Function in a Complementary Manner to Increase Pattern-Recognition Receptor-Induced Outcomes in Human MacrophagesMatija Hedl, Rui Sun, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|August 6, 2021
The E3 ubiquitin ligase RNF186 and <i>RNF186</i> risk variants regulate innate receptor-induced outcomesKishu Ranjan, Matija Hedl, Clara Abraham
Gut|May 18, 2019
IL23 induces IL23R recycling and amplifies innate receptor-induced signalling and cytokines in human macrophages, and the IBD-protective IL23R R381Q variant modulates these outcomesRui Sun, Matija Hedl, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|May 25, 2011
Inflammatory disease protective R381Q IL23 receptor polymorphism results in decreased primary CD4+ and CD8+ human T-cell functional responsesRitu Sarin, Xingxin Wu, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|September 25, 2016
JAK2 Disease-Risk Variants Are Gain of Function and JAK Signaling Threshold Determines Innate Receptor-Induced Proinflammatory Cytokine Secretion in MacrophagesMatija Hedl, Deborah D Proctor, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|February 6, 2007
CD18 is required for intestinal T cell responses at multiple immune checkpointsMarissa Marski, Alice L Ye, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|December 30, 2018
IRF5 Is Required for Bacterial Clearance in Human M1-Polarized Macrophages, and <i>IRF5</i> Immune-Mediated Disease Risk Variants Modulate This OutcomeMatija Hedl, Jie Yan, Heiko Witt, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 17, 2019
STAT3 and STAT5 Signaling Thresholds Determine Distinct Regulation for Innate Receptor-Induced Inflammatory Cytokines, and <i>STAT3</i>/<i>STAT5</i> Disease Variants Modulate These OutcomesMatija Hedl, Rui Sun, Chen Huang, et al.
BMC Medical Genomics|June 30, 2010
Pathway analysis comparison using Crohn's disease genome wide association studiesDavid Ballard, Clara Abraham, Judy Cho, et al.
Cell Reports|December 26, 2019
LACC1 Required for NOD2-Induced, ER Stress-Mediated Innate Immune Outcomes in Human Macrophages and LACC1 Risk Variants Modulate These OutcomesChen Huang, Matija Hedl, Kishu Ranjan, et al.
Pageof 9

Showing results (31-40 of 87) with videos related to

Sort By:
Pageof 9
Journal of Immunology (Baltimore, Md. : 1950)|July 15, 2020
Disease Risk-Associated Genetic Variants in <i>STAT1</i> and <i>STAT4</i> Function in a Complementary Manner to Increase Pattern-Recognition Receptor-Induced Outcomes in Human MacrophagesMatija Hedl, Rui Sun, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|August 6, 2021
The E3 ubiquitin ligase RNF186 and <i>RNF186</i> risk variants regulate innate receptor-induced outcomesKishu Ranjan, Matija Hedl, Clara Abraham
Gut|May 18, 2019
IL23 induces IL23R recycling and amplifies innate receptor-induced signalling and cytokines in human macrophages, and the IBD-protective IL23R R381Q variant modulates these outcomesRui Sun, Matija Hedl, Clara Abraham
Proceedings of the National Academy of Sciences of the United States of America|May 25, 2011
Inflammatory disease protective R381Q IL23 receptor polymorphism results in decreased primary CD4+ and CD8+ human T-cell functional responsesRitu Sarin, Xingxin Wu, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|September 25, 2016
JAK2 Disease-Risk Variants Are Gain of Function and JAK Signaling Threshold Determines Innate Receptor-Induced Proinflammatory Cytokine Secretion in MacrophagesMatija Hedl, Deborah D Proctor, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|February 6, 2007
CD18 is required for intestinal T cell responses at multiple immune checkpointsMarissa Marski, Alice L Ye, Clara Abraham
Journal of Immunology (Baltimore, Md. : 1950)|December 30, 2018
IRF5 Is Required for Bacterial Clearance in Human M1-Polarized Macrophages, and <i>IRF5</i> Immune-Mediated Disease Risk Variants Modulate This OutcomeMatija Hedl, Jie Yan, Heiko Witt, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 17, 2019
STAT3 and STAT5 Signaling Thresholds Determine Distinct Regulation for Innate Receptor-Induced Inflammatory Cytokines, and <i>STAT3</i>/<i>STAT5</i> Disease Variants Modulate These OutcomesMatija Hedl, Rui Sun, Chen Huang, et al.
BMC Medical Genomics|June 30, 2010
Pathway analysis comparison using Crohn's disease genome wide association studiesDavid Ballard, Clara Abraham, Judy Cho, et al.
Cell Reports|December 26, 2019
LACC1 Required for NOD2-Induced, ER Stress-Mediated Innate Immune Outcomes in Human Macrophages and LACC1 Risk Variants Modulate These OutcomesChen Huang, Matija Hedl, Kishu Ranjan, et al.
Pageof 9