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Clara Soria-Valles

Showing results (1-10 of 17) with videos related to

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Trends in Molecular Medicine|June 12, 2016
iPSCs: On the Road to Reprogramming AgingClara Soria-Valles, Carlos López-Otín
Disease Models & Mechanisms|August 3, 2016
Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cellsDido Carrero, Clara Soria-Valles, Carlos López-Otín
Cell Cycle (Georgetown, Tex.)|September 17, 2015
Reprogramming aging through DOT1L inhibitionClara Soria-Valles, Fernando G Osorio, Carlos López-Otín
Experimental Hematology|October 23, 2012
Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasiaTeresa Bernal, Angela Moncada-Pazos, Clara Soria-Valles, et al.
Mechanisms of Ageing and Development|October 11, 2016
Immune and inflammatory responses to DNA damage in cancer and agingClara Soria-Valles, Alejandro López-Soto, Fernando G Osorio, et al.
Human Mutation|May 28, 2011
Analysis of the disintegrin-metalloproteinases family reveals ADAM29 and ADAM7 are often mutated in melanomaXiaomu Wei, Angela Moncada-Pazos, Santiago Cal, et al.
Genes & Development|September 29, 2012
Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory responseFernando G Osorio, Clea Bárcena, Clara Soria-Valles, et al.
Journal of Andrology|October 22, 2008
Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamstersAna Coto-Montes, Cristina Tomás-Zapico, Jorge Martínez-Fraga, et al.
The EMBO Journal|May 21, 2015
Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acidAna Gutiérrez-Fernández, Clara Soria-Valles, Fernando G Osorio, et al.
Journal of Pineal Research|December 19, 2008
Melatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated miceBeatriz Caballero, Ignacio Vega-Naredo, Verónica Sierra, et al.
Pageof 2

Showing results (1-10 of 17) with videos related to

Sort By:
Pageof 2
Trends in Molecular Medicine|June 12, 2016
iPSCs: On the Road to Reprogramming AgingClara Soria-Valles, Carlos López-Otín
Disease Models & Mechanisms|August 3, 2016
Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cellsDido Carrero, Clara Soria-Valles, Carlos López-Otín
Cell Cycle (Georgetown, Tex.)|September 17, 2015
Reprogramming aging through DOT1L inhibitionClara Soria-Valles, Fernando G Osorio, Carlos López-Otín
Experimental Hematology|October 23, 2012
Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasiaTeresa Bernal, Angela Moncada-Pazos, Clara Soria-Valles, et al.
Mechanisms of Ageing and Development|October 11, 2016
Immune and inflammatory responses to DNA damage in cancer and agingClara Soria-Valles, Alejandro López-Soto, Fernando G Osorio, et al.
Human Mutation|May 28, 2011
Analysis of the disintegrin-metalloproteinases family reveals ADAM29 and ADAM7 are often mutated in melanomaXiaomu Wei, Angela Moncada-Pazos, Santiago Cal, et al.
Genes & Development|September 29, 2012
Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory responseFernando G Osorio, Clea Bárcena, Clara Soria-Valles, et al.
Journal of Andrology|October 22, 2008
Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamstersAna Coto-Montes, Cristina Tomás-Zapico, Jorge Martínez-Fraga, et al.
The EMBO Journal|May 21, 2015
Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acidAna Gutiérrez-Fernández, Clara Soria-Valles, Fernando G Osorio, et al.
Journal of Pineal Research|December 19, 2008
Melatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated miceBeatriz Caballero, Ignacio Vega-Naredo, Verónica Sierra, et al.
Pageof 2