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Circulation. Heart Failure|January 1, 2015
Tumor necrosis factor: a mechanistic link between angiotensin-II-induced cardiac inflammation and fibrosisClemens Duerrschmid, JoAnn Trial, Yanlin Wang, et al.Frontiers in Immunology|September 26, 2013
Th1/M1 conversion to th2/m2 responses in models of inflammation lacking cell death stimulates maturation of monocyte precursors to fibroblastsJoann Trial, Katarzyna A Cieslik, Sandra B Haudek, et al.Science Advances|February 22, 2023
Asprosin promotes feeding through SK channel-dependent activation of AgRP neuronsBing Feng, Hesong Liu, Ila Mishra, et al.Physiological Reports|April 30, 2016
TNF/Ang-II synergy is obligate for fibroinflammatory pathology, but not for changes in cardiorenal functionMagdalena Mayr, Clemens Duerrschmid, Guillermo Medrano, et al.Journal of Molecular and Cellular Cardiology|January 23, 2013
TNF receptor 1 signaling is critically involved in mediating angiotensin-II-induced cardiac fibrosisClemens Duerrschmid, Jeffrey R Crawford, Erin Reineke, et al.Elife|April 27, 2021
Asprosin-neutralizing antibodies as a treatment for metabolic syndromeIla Mishra, Clemens Duerrschmid, Zhiqiang Ku, et al.Cell|April 19, 2016
Asprosin, a Fasting-Induced Glucogenic Protein HormoneChase Romere, Clemens Duerrschmid, Juan Bournat, et al.Nature Medicine|November 7, 2017
Asprosin is a centrally acting orexigenic hormoneClemens Duerrschmid, Yanlin He, Chunmei Wang, et al.Pageof 1