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Proceedings of the National Academy of Sciences of the United States of America|February 1, 1993
Redundant cyclin overexpression and gene amplification in breast cancer cellsK Keyomarsi, A B PardeeOncogene|November 30, 1999
UCN-01-mediated G1 arrest in normal but not tumor breast cells is pRb-dependent and p53-independentX Chen, M Lowe, K KeyomarsiAdvances in Experimental Medicine and Biology|January 1, 1988
Tumor cell responses to inhibition of thymidylate synthaseR G Moran, K Keyomarsi, R PatelCancer Research|February 15, 1997
Lovastatin induction of cyclin-dependent kinase inhibitors in human breast cells occurs in a cell cycle-independent fashionJ Gray-Bablin, S Rao, K KeyomarsiOncogene|June 30, 2009
Post-translational modification and stability of low molecular weight cyclin EB B Mull, J Cox, T Bui, et al.Oncogene|November 12, 1998
Lovastatin mediated G1 arrest in normal and tumor breast cells is through inhibition of CDK2 activity and redistribution of p21 and p27, independent of p53S Rao, M Lowe, T W Herliczek, et al.Oncogene|September 7, 1995
Deregulation of cyclin E in breast cancerK Keyomarsi, D Conte, W Toyofuku, et al.The Journal of Biological Chemistry|July 15, 1993
The thymidylate synthase inhibitor, ICI D1694, overcomes translational detainment of the enzymeK Keyomarsi, J Samet, G Molnar, et al.The Journal of Biological Chemistry|October 5, 1992
Cloning and characterization of human cyclin D3, a cDNA closely related in sequence to the PRAD1/cyclin D1 proto-oncogeneT Motokura, K Keyomarsi, H M Kronenberg, et al.Cancer Research|July 1, 1991
Synchronization of tumor and normal cells from G1 to multiple cell cycles by lovastatinK Keyomarsi, L Sandoval, V Band, et al.Pageof 8