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Dale Abel

Showing results (61-70 of 266) with videos related to

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Endocrine Journal|September 13, 2011
Conditional deletion of insulin receptor in thyrocytes does not affect thyroid structure and functionSangmi Ock, Seok Hong Lee, Jihyun Ahn, et al.
Journal of Molecular and Cellular Cardiology|October 9, 2017
Activation of IGF-1 receptors and Akt signaling by systemic hyperinsulinemia contributes to cardiac hypertrophy but does not regulate cardiac autophagy in obese diabetic miceKarla Maria Pires, Marcio Buffolo, Christin Schaaf, et al.
IEEE Transactions on Bio-Medical Engineering|August 24, 2011
The maximal downstroke of epicardial potentials as an index of electrical activity in mouse heartsKwanghyun Sohn, Frank B Sachse, Alonso P Moreno, et al.
The FEBS Journal|January 7, 2012
Cytosolic, but not mitochondrial, oxidative stress is a likely contributor to cardiac hypertrophy resulting from cardiac specific GLUT4 deletion in miceYan Li, Adam R Wende, Orathai Nunthakungwan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 27, 2019
Increased glycolysis mediates Wnt7b-induced bone formationHong Chen, Xing Ji, Wen-Chih Lee, et al.
Journal of Molecular and Cellular Cardiology Plus|September 26, 2024
Comparative analysis of two independent Myh6-Cre transgenic mouse linesAmanda Davenport, Chase W Kessinger, Ryan D Pfeiffer, et al.
Cardiovascular Research|January 25, 2005
Erythropoietin prevents the acute myocardial inflammatory response induced by ischemia/reperfusion via induction of AP-1Tao Rui, Qingping Feng, Ming Lei, et al.
Journal of the American Heart Association|September 21, 2013
Inducible overexpression of GLUT1 prevents mitochondrial dysfunction and attenuates structural remodeling in pressure overload but does not prevent left ventricular dysfunctionRenata O Pereira, Adam R Wende, Curtis Olsen, et al.
Journal of Molecular and Cellular Cardiology|March 3, 2009
Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarctionSandra Sena, Ping Hu, Dongfang Zhang, et al.
Circulation. Heart Failure|December 10, 2013
Deep sequence analysis of gene expression identifies osteopontin as a downstream effector of integrin-linked kinase (ILK) in cardiac-specific ILK knockout miceJing Dai, Takashi Matsui, E Dale Abel, et al.
Pageof 27

Showing results (61-70 of 266) with videos related to

Sort By:
Pageof 27
Endocrine Journal|September 13, 2011
Conditional deletion of insulin receptor in thyrocytes does not affect thyroid structure and functionSangmi Ock, Seok Hong Lee, Jihyun Ahn, et al.
Journal of Molecular and Cellular Cardiology|October 9, 2017
Activation of IGF-1 receptors and Akt signaling by systemic hyperinsulinemia contributes to cardiac hypertrophy but does not regulate cardiac autophagy in obese diabetic miceKarla Maria Pires, Marcio Buffolo, Christin Schaaf, et al.
IEEE Transactions on Bio-Medical Engineering|August 24, 2011
The maximal downstroke of epicardial potentials as an index of electrical activity in mouse heartsKwanghyun Sohn, Frank B Sachse, Alonso P Moreno, et al.
The FEBS Journal|January 7, 2012
Cytosolic, but not mitochondrial, oxidative stress is a likely contributor to cardiac hypertrophy resulting from cardiac specific GLUT4 deletion in miceYan Li, Adam R Wende, Orathai Nunthakungwan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 27, 2019
Increased glycolysis mediates Wnt7b-induced bone formationHong Chen, Xing Ji, Wen-Chih Lee, et al.
Journal of Molecular and Cellular Cardiology Plus|September 26, 2024
Comparative analysis of two independent Myh6-Cre transgenic mouse linesAmanda Davenport, Chase W Kessinger, Ryan D Pfeiffer, et al.
Cardiovascular Research|January 25, 2005
Erythropoietin prevents the acute myocardial inflammatory response induced by ischemia/reperfusion via induction of AP-1Tao Rui, Qingping Feng, Ming Lei, et al.
Journal of the American Heart Association|September 21, 2013
Inducible overexpression of GLUT1 prevents mitochondrial dysfunction and attenuates structural remodeling in pressure overload but does not prevent left ventricular dysfunctionRenata O Pereira, Adam R Wende, Curtis Olsen, et al.
Journal of Molecular and Cellular Cardiology|March 3, 2009
Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarctionSandra Sena, Ping Hu, Dongfang Zhang, et al.
Circulation. Heart Failure|December 10, 2013
Deep sequence analysis of gene expression identifies osteopontin as a downstream effector of integrin-linked kinase (ILK) in cardiac-specific ILK knockout miceJing Dai, Takashi Matsui, E Dale Abel, et al.
Pageof 27