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Daniel C Bartos

Showing results (1-10 of 16) with videos related to

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Comprehensive Physiology|July 4, 2015
Ion Channels in the HeartDaniel C Bartos, Eleonora Grandi, Crystal M Ripplinger
Biophysical Journal|September 18, 2014
Depolarization of cardiac membrane potential synchronizes calcium sparks and waves in tissueDaisuke Sato, Daniel C Bartos, Kenneth S Ginsburg, et al.
The Journal of Physiology|December 24, 2016
Quantitative analysis of the Ca<sup>2+</sup> -dependent regulation of delayed rectifier K<sup>+</sup> current I<sub>Ks</sub> in rabbit ventricular myocytesDaniel C Bartos, Stefano Morotti, Kenneth S Ginsburg, et al.
Mayo Clinic Proceedings|November 5, 2016
The Promise and Peril of Precision Medicine: Phenotyping Still Matters MostJaeger P Ackerman, Daniel C Bartos, Jamie D Kapplinger, et al.
American Journal of Physiology. Cell Physiology|April 15, 2011
Trafficking-deficient hERG K⁺ channels linked to long QT syndrome are regulated by a microtubule-dependent quality control compartment in the ERJennifer L Smith, Christie M McBride, Parvathi S Nataraj, et al.
Heart Rhythm|November 26, 2013
A KCNQ1 mutation contributes to the concealed type 1 long QT phenotype by limiting the Kv7.1 channel conformational changes associated with protein kinase A phosphorylationDaniel C Bartos, John R Giudicessi, David J Tester, et al.
American Journal of Physiology. Cell Physiology|February 1, 2013
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibilityElizabeth A Schroder, Mellani Lefta, Xiping Zhang, et al.
Heart Rhythm|February 22, 2015
The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarizationElizabeth A Schroder, Don E Burgess, Xiping Zhang, et al.
International Journal of Molecular Sciences|July 9, 2022
Mutation-Specific Differences in Kv7.1 (<i>KCNQ1</i>) and Kv11.1 (<i>KCNH2</i>) Channel Dysfunction and Long QT Syndrome PhenotypesPeter M Kekenes-Huskey, Don E Burgess, Bin Sun, et al.
The Journal of Membrane Biology|April 3, 2013
Mechanistic basis for type 2 long QT syndrome caused by KCNH2 mutations that disrupt conserved arginine residues in the voltage sensorChristie M McBride, Ashley M Smith, Jennifer L Smith, et al.
Pageof 2

Showing results (1-10 of 16) with videos related to

Sort By:
Pageof 2
Comprehensive Physiology|July 4, 2015
Ion Channels in the HeartDaniel C Bartos, Eleonora Grandi, Crystal M Ripplinger
Biophysical Journal|September 18, 2014
Depolarization of cardiac membrane potential synchronizes calcium sparks and waves in tissueDaisuke Sato, Daniel C Bartos, Kenneth S Ginsburg, et al.
The Journal of Physiology|December 24, 2016
Quantitative analysis of the Ca<sup>2+</sup> -dependent regulation of delayed rectifier K<sup>+</sup> current I<sub>Ks</sub> in rabbit ventricular myocytesDaniel C Bartos, Stefano Morotti, Kenneth S Ginsburg, et al.
Mayo Clinic Proceedings|November 5, 2016
The Promise and Peril of Precision Medicine: Phenotyping Still Matters MostJaeger P Ackerman, Daniel C Bartos, Jamie D Kapplinger, et al.
American Journal of Physiology. Cell Physiology|April 15, 2011
Trafficking-deficient hERG K⁺ channels linked to long QT syndrome are regulated by a microtubule-dependent quality control compartment in the ERJennifer L Smith, Christie M McBride, Parvathi S Nataraj, et al.
Heart Rhythm|November 26, 2013
A KCNQ1 mutation contributes to the concealed type 1 long QT phenotype by limiting the Kv7.1 channel conformational changes associated with protein kinase A phosphorylationDaniel C Bartos, John R Giudicessi, David J Tester, et al.
American Journal of Physiology. Cell Physiology|February 1, 2013
The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibilityElizabeth A Schroder, Mellani Lefta, Xiping Zhang, et al.
Heart Rhythm|February 22, 2015
The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarizationElizabeth A Schroder, Don E Burgess, Xiping Zhang, et al.
International Journal of Molecular Sciences|July 9, 2022
Mutation-Specific Differences in Kv7.1 (<i>KCNQ1</i>) and Kv11.1 (<i>KCNH2</i>) Channel Dysfunction and Long QT Syndrome PhenotypesPeter M Kekenes-Huskey, Don E Burgess, Bin Sun, et al.
The Journal of Membrane Biology|April 3, 2013
Mechanistic basis for type 2 long QT syndrome caused by KCNH2 mutations that disrupt conserved arginine residues in the voltage sensorChristie M McBride, Ashley M Smith, Jennifer L Smith, et al.
Pageof 2