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Biorxiv : the Preprint Server for Biology|January 7, 2025
The CD4 T cell-independent IgG response during persistent virus infection favors emergence of neutralization-escape variantsKatelyn N Ayers, Matthew D Lauver, Kalynn M Alexander, et al.Proceedings of the National Academy of Sciences of the United States of America|June 2, 2021
High-resolution asymmetric structure of a Fab-virus complex reveals overlap with the receptor binding siteDaniel J Goetschius, Samantha R Hartmann, Lindsey J Organtini, et al.Elife|September 17, 2020
Antibody escape by polyomavirus capsid mutation facilitates neurovirulenceMatthew D Lauver, Daniel J Goetschius, Colleen S Netherby-Winslow, et al.Nature Communications|October 10, 2023
A human monoclonal antibody binds within the poliovirus receptor-binding site to neutralize all three serotypesAndrew J Charnesky, Julia E Faust, Hyunwook Lee, et al.Journal of Virology|May 18, 2023
Viral Capsid, Antibody, and Receptor Interactions: Experimental Analysis of the Antibody Escape Evolution of Canine ParvovirusRobert A López-Astacio, Oluwafemi F Adu, Daniel J Goetschius, et al.Biorxiv : the Preprint Server for Biology|January 30, 2023
Viral capsid, antibody, and receptor interactions: experimental analysis of the antibody escape evolution of canine parvovirusRobert A López-Astacio, Oluwafemi F Adu, Daniel J Goetschius, et al.Pageof 2