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Experimental Neurology|November 29, 2005
Morris water maze search strategy analysis in PDAPP mice before and after experimental traumatic brain injuryDavid L Brody, David M HoltzmanAnnual Review of Neuroscience|March 21, 2008
Active and passive immunotherapy for neurodegenerative disordersDavid L Brody, David M HoltzmanThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 1, 2011
Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalitiesHien T Tran, Frank M LaFerla, David M Holtzman, et al.Neurobiology of Disease|August 5, 2010
Traumatic brain injury reduces soluble extracellular amyloid-β in mice: a methodologically novel combined microdialysis-controlled cortical impact studyKatherine E Schwetye, John R Cirrito, Thomas J Esparza, et al.Annals of Neurology|December 11, 2012
Amyloid-β oligomerization in Alzheimer dementia versus high-pathology controlsThomas J Esparza, Hanzhi Zhao, John R Cirrito, et al.Brain : a Journal of Neurology|November 26, 2011
Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injurySandra Magnoni, Thomas J Esparza, Valeria Conte, et al.Science (New York, N.Y.)|August 30, 2008
Amyloid-beta dynamics correlate with neurological status in the injured human brainDavid L Brody, Sandra Magnoni, Kate E Schwetye, et al.Neurobiology of Aging|November 15, 2011
CSF biomarkers for Alzheimer's disease: current utility and potential future useDavid M HoltzmanEMBO Molecular Medicine|January 6, 2010
A surrogate marker for Abeta42 production in the CNSDavid M HoltzmanJournal of Molecular Neuroscience : MN|June 8, 2004
In vivo effects of ApoE and clusterin on amyloid-beta metabolism and neuropathologyDavid M HoltzmanPageof 65