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Toxicology
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February 25, 2017
Models of toxicity of diacetyl and alternative diones
David M Brass, Scott M Palmer
Seminars in Respiratory and Critical Care Medicine
|
February 18, 2009
Host-environment interactions in exposure-related diffuse lung diseases
David M Brass, Anastasia L Wise, David A Schwartz
Proceedings of the American Thoracic Society
|
January 5, 2007
Using mouse genomics to understand idiopathic interstitial fibrosis
David M Brass, John Tomfohr, Ivana V Yang, et al.
La Medicina Del Lavoro
|
July 7, 2020
Occupational asthma and rhinitis due to wheat flour: sublingual specific immunotherapy treatment
Marco Dubini, Paolo Marraccini, David M Brass, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
June 24, 2003
Fibrinolysis in LPS-induced chronic airway disease
Jordan D Savov, David M Brass, Katherine G Berman, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
October 12, 2002
Neutrophils play a critical role in development of LPS-induced airway disease
Jordan D Savov, Stephen H Gavett, David M Brass, et al.
The Journal of Allergy and Clinical Immunology
|
September 10, 2004
LPS binding protein is important in the airway response to inhaled endotoxin
David M Brass, Jordan D Savov, Gregory S Whitehead, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
March 27, 2007
CD14 is an essential mediator of LPS-induced airway disease
David M Brass, John W Hollingsworth, Erin McElvania-Tekippe, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
April 19, 2005
Toll-like receptor 4 antagonist (E5564) prevents the chronic airway response to inhaled lipopolysaccharide
Jordan D Savov, David M Brass, Barbara L Lawson, et al.
American Journal of Respiratory Cell and Molecular Biology
|
June 10, 2008
Chronic LPS inhalation causes emphysema-like changes in mouse lung that are associated with apoptosis
David M Brass, John W Hollingsworth, Mark Cinque, et al.
Page
of 4
Search research articles
Search
Showing results (1-10 of 33) with videos related to
Sort By:
Page
of 4
Toxicology
|
February 25, 2017
Models of toxicity of diacetyl and alternative diones
David M Brass, Scott M Palmer
Seminars in Respiratory and Critical Care Medicine
|
February 18, 2009
Host-environment interactions in exposure-related diffuse lung diseases
David M Brass, Anastasia L Wise, David A Schwartz
Proceedings of the American Thoracic Society
|
January 5, 2007
Using mouse genomics to understand idiopathic interstitial fibrosis
David M Brass, John Tomfohr, Ivana V Yang, et al.
La Medicina Del Lavoro
|
July 7, 2020
Occupational asthma and rhinitis due to wheat flour: sublingual specific immunotherapy treatment
Marco Dubini, Paolo Marraccini, David M Brass, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
June 24, 2003
Fibrinolysis in LPS-induced chronic airway disease
Jordan D Savov, David M Brass, Katherine G Berman, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
October 12, 2002
Neutrophils play a critical role in development of LPS-induced airway disease
Jordan D Savov, Stephen H Gavett, David M Brass, et al.
The Journal of Allergy and Clinical Immunology
|
September 10, 2004
LPS binding protein is important in the airway response to inhaled endotoxin
David M Brass, Jordan D Savov, Gregory S Whitehead, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
March 27, 2007
CD14 is an essential mediator of LPS-induced airway disease
David M Brass, John W Hollingsworth, Erin McElvania-Tekippe, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology
|
April 19, 2005
Toll-like receptor 4 antagonist (E5564) prevents the chronic airway response to inhaled lipopolysaccharide
Jordan D Savov, David M Brass, Barbara L Lawson, et al.
American Journal of Respiratory Cell and Molecular Biology
|
June 10, 2008
Chronic LPS inhalation causes emphysema-like changes in mouse lung that are associated with apoptosis
David M Brass, John W Hollingsworth, Mark Cinque, et al.
Page
of 4