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Scientific Reports|January 18, 2018
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entryJustyna Broniarczyk, Nadja Ring, Paola Massimi, et al.
Journal of Virology|May 27, 2026
Rethinking residual p53 function in HPV-positive cervical cancer cellsSannia Farrukh, Nezka Kavcic, Giulia Canarutto, et al.
Gene|August 30, 2008
Cloning and functional analysis of the promoter region of the human Disc large geneAna Laura Cavatorta, Adriana A Giri, Lawrence Banks, et al.
Biological Chemistry|November 3, 2007
Cellular localization of MAGI-1 caspase cleavage products and their role in apoptosisSaska Ivanova, Urska Repnik, Lawrence Banks, et al.
Journal of Virology|December 16, 2014
Characterization of novel transcripts of human papillomavirus type 16 using cap analysis gene expression technologyAyumi Taguchi, Kazunori Nagasaka, Kei Kawana, et al.
Journal of Virology|October 5, 2022
Repression of Memo1, a Novel Target of Human Papillomavirus Type 16 E7, Increases Cell Proliferation in Cervical Cancer CellsOscar Trejo-Cerro, Paola Massimi, Justyna Broniarczyk, et al.
Tumour Virus Research|September 2, 2023
Identification and characterisation of novel potential phospho-acceptor sites in HPV-16 E7Oscar Trejo-Cerro, Justyna Broniarczyk, Nezka Kavcic, et al.
Nucleic Acids Research|December 5, 2003
Repair of a minimal DNA double-strand break by NHEJ requires DNA-PKcs and is controlled by the ATM/ATR checkpointChristian Kühne, Marie-Louise Tjörnhammar, Sándor Pongor, et al.
Current Pharmaceutical Design|May 30, 2013
Disordered interactome of human papillomavirusBin Xue, Ketaki Ganti, Alejandro Rabionet, et al.
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