Showing results (11-20 of 171) with videos related to
Sort By:
Pageof 18
Journal of Virology|October 31, 2014
PDZRN3/LNX3 is a novel target of human papillomavirus type 16 (HPV-16) and HPV-18 E6Miranda Thomas, Lawrence BanksCurrent Opinion in Virology|October 16, 2021
The biology of papillomavirus PDZ associations: what do they offer papillomaviruses?Miranda Thomas, Lawrence BanksVirology|August 25, 2009
The stability of the human papillomavirus E6 oncoprotein is E6AP dependentVjekoslav Tomaić, David Pim, Lawrence BanksInternational Journal of Cancer|June 2, 2004
HPV-18 E6*I modulates HPV-18 full-length E6 functions in a cell cycle dependent mannerErnesto Guccione, David Pim, Lawrence BanksJournal of Virology|July 31, 2009
The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 proteinDavid Pim, Vjekoslav Tomaic, Lawrence BanksCurrent Opinion in Cell Biology|May 26, 2019
Choosing the right path: membrane trafficking and infectious entry of small DNA tumor virusesMarina Bugnon Valdano, David Pim, Lawrence BanksFrontiers in Cellular and Infection Microbiology|June 14, 2019
Diverse Papillomavirus Types Induce Endosomal TubulationAbida Siddiqa, Paola Massimi, David Pim, et al.Oncogene|July 27, 2005
Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2ADavid Pim, Paola Massimi, Stephen M Dilworth, et al.Mbio|October 18, 2022
HPV-16 E7 Interacts with the Endocytic Machinery via the AP2 Adaptor μ2 SubunitOm Basukala, Oscar Trejo-Cerro, Michael P Myers, et al.Oncogene|September 21, 2004
HPV E6 specifically targets different cellular pools of its PDZ domain-containing tumour suppressor substrates for proteasome-mediated degradationPaola Massimi, Noor Gammoh, Miranda Thomas, et al.Pageof 18