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The Journal of Biological Chemistry|March 14, 2008
Role of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injuryNaoko Hanawa, Mie Shinohara, Behnam Saberi, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 24, 2025
Basic Science and PathogenesisBrian Carson, Josephine Chu, Devaraj V Chandrashekar, et al.
Methods in Enzymology|June 2, 2010
Determination of GSH, GSSG, and GSNO using HPLC with electrochemical detectionLi-Peng Yap, Harsh Sancheti, Maria D Ybanez, et al.
Chemical Research in Toxicology|April 11, 2014
Respiratory substrates regulate S-nitrosylation of mitochondrial proteins through a thiol-dependent pathwayAllen H K Chang, Harsh Sancheti, Jerome Garcia, et al.
Hepatology (Baltimore, Md.)|June 17, 2015
Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosisLily Dara, Heather Johnson, Jo Suda, et al.
Gastroenterology|July 13, 2006
c-Jun N-terminal kinase plays a major role in murine acetaminophen hepatotoxicityBasuki K Gunawan, Zhang-Xu Liu, Derick Han, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 23, 2025
Basic Science and PathogenesisOghenetega E Imiruaye, Isis Perez, Brian Carson, et al.
Plos One|May 25, 2022
Comparative studies between the murine immortalized brain endothelial cell line (bEnd.3) and induced pluripotent stem cell-derived human brain endothelial cells for paracellular transportJiahong Sun, Weijun Ou, Derick Han, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 23, 2025
Basic Science and PathogenesisJosephine Chu, Ross A Steinberg, Brian Carson, et al.
Hepatology (Baltimore, Md.)|July 23, 2013
Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through c-jun-N-terminal kinase (JNK)-dependent and -independent signaling pathwaysBehnam Saberi, Maria D Ybanez, Heather S Johnson, et al.
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