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Cell Metabolism|December 7, 2007
FoxO3 controls dangerous proteolytic liaisonsDidier Attaix, Daniel BechetCell Metabolism|November 13, 2012
The missing link: Mul1 signals mitophagy and muscle wastingDidier Attaix, Daniel TaillandierCurrent Opinion in Rheumatology|October 21, 2006
Mechanisms of skeletal muscle atrophySophie Ventadour, Didier AttaixFrontiers in Physiology|March 26, 2015
Role of E2-Ub-conjugating enzymes during skeletal muscle atrophyCecile Polge, Didier Attaix, Daniel TaillandierJournal of the American Society of Nephrology : JASN|January 17, 2002
Insights into the abnormalities of chronic renal disease attributed to malnutritionWilliam E MitchKidney International. Supplement|March 9, 2005
Beneficial responses to modified diets in treating patients with chronic kidney diseaseWilliam E MitchThe Journal of Clinical Investigation|June 3, 2005
Cachexia in chronic kidney disease: a link to defective central nervous system control of appetiteWilliam E MitchJournal of Renal Nutrition : the Official Journal of the Council on Renal Nutrition of the National Kidney Foundation|January 3, 2007
Malnutrition is an unusual cause of decreased muscle mass in chronic kidney diseaseWilliam E MitchJournal of Renal Nutrition : the Official Journal of the Council on Renal Nutrition of the National Kidney Foundation|July 11, 2006
Proteolytic mechanisms, not malnutrition, cause loss of muscle mass in kidney failureWilliam E MitchCurrent Opinion in Supportive and Palliative Care|December 17, 2008
Role of the ubiquitin-proteasome pathway in muscle atrophy in cachexiaDidier Attaix, Lydie Combaret, Daniel Béchet, et al.Pageof 17