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Cancer Discovery|January 20, 2022
RAS at 40: Update from the RAS InitiativeDwight V Nissley, Frank McCormickCell|July 1, 2017
RAS Proteins and Their Regulators in Human DiseaseDhirendra K Simanshu, Dwight V Nissley, Frank McCormickScientific Reports|October 25, 2024
The prominent pervasive oncogenic role and tissue specific permissiveness of RAS gene mutationsMing Yi, Daniel Soppet, Frank McCormick, et al.Methods in Molecular Biology (Clifton, N.J.)|April 3, 2024
Progress in Targeting KRAS DirectlyDwight V Nissley, Andrew G Stephen, Ming Yi, et al.Scientific Reports|June 26, 2020
ssGSEA score-based Ras dependency indexes derived from gene expression data reveal potential Ras addiction mechanisms with possible clinical implicationsMing Yi, Dwight V Nissley, Frank McCormick, et al.Cancer Informatics|June 22, 2017
Tumor RAS Gene Expression Levels Are Influenced by the Mutational Status of RAS Genes and Both Upstream and Downstream RAS Pathway GenesRobert M Stephens, Ming Yi, Bailey Kessing, et al.Biomolecular NMR Assignments|May 2, 2025
NMR 1H, 13C, and 15N resonance assignments of the oncogenic Q61R variant of human NRAS in the active, GTP-bound conformationAlok K Sharma, Marco Tonelli, Marcin Dyba, et al.Proceedings of the National Academy of Sciences of the United States of America|January 29, 2022
Classical RAS proteins are not essential for paradoxical ERK activation induced by RAF inhibitorsLick Pui Lai, Nicole Fer, William Burgan, et al.Biomolecular NMR Assignments|October 23, 2021
NMR 1H, 13C, 15N backbone resonance assignments of the T35S and oncogenic T35S/Q61L mutants of human KRAS4b in the active, GppNHp-bound conformationAlok K Sharma, Marcin Dyba, Marco Tonelli, et al.Cell Reports|July 23, 2020
Structural Insights into the SPRED1-Neurofibromin-KRAS Complex and Disruption of SPRED1-Neurofibromin Interaction by Oncogenic EGFRWupeng Yan, Evan Markegard, Srisathiyanarayanan Dharmaiah, et al.Pageof 32