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E Braggio

Showing results (1-10 of 12) with videos related to

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Leukemia|February 16, 2012
Absence of tissue factor expression by neoplastic plasma cells in multiple myelomaG Cesarman-Maus, E Braggio, H Maldonado, et al.
Blood Cancer Journal|July 23, 2013
Lessons from next-generation sequencing analysis in hematological malignanciesE Braggio, J B Egan, R Fonseca, et al.
Brazilian Journal of Medical and Biological Research = Revista Brasileira De Pesquisas Medicas E Biologicas|April 14, 2016
Genetic aberrations in multiple myeloma characterized by cIg-FISH: a Brazilian contextP Segges, E Braggio, C Minnicelli, et al.
Journal of Clinical Pathology|May 29, 2004
Identification of three novel RB1 mutations in Brazilian patients with retinoblastoma by "exon by exon" PCR mediated SSCP analysisE Braggio, C R Bonvicino, F R Vargas, et al.
Blood Cancer Journal|September 23, 2017
High somatic mutation and neoantigen burden are correlated with decreased progression-free survival in multiple myelomaA Miller, Y Asmann, L Cattaneo, et al.
Leukemia|July 19, 2014
Monoclonal B-cell lymphocytosis is characterized by mutations in CLL putative driver genes and clonal heterogeneity many years before disease progressionJ Ojha, C Secreto, K Rabe, et al.
Leukemia|July 30, 2016
Genomic characterization of high-count MBL cases indicates that early detection of driver mutations and subclonal expansion are predictors of adverse clinical outcomeS Barrio, T D Shanafelt, J Ojha, et al.
Leukemia|January 21, 2012
Longitudinal genome-wide analysis of patients with chronic lymphocytic leukemia reveals complex evolution of clonal architecture at disease progression and at the time of relapseE Braggio, N E Kay, S VanWier, et al.
Blood Cancer Journal|February 27, 2016
Panel sequencing for clinically oriented variant screening and copy number detection in 142 untreated multiple myeloma patientsK M Kortuem, E Braggio, L Bruins, et al.
Leukemia|June 12, 2013
Genome-wide studies in multiple myeloma identify XPO1/CRM1 as a critical target validated using the selective nuclear export inhibitor KPT-276J Schmidt, E Braggio, K M Kortuem, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Leukemia|February 16, 2012
Absence of tissue factor expression by neoplastic plasma cells in multiple myelomaG Cesarman-Maus, E Braggio, H Maldonado, et al.
Blood Cancer Journal|July 23, 2013
Lessons from next-generation sequencing analysis in hematological malignanciesE Braggio, J B Egan, R Fonseca, et al.
Brazilian Journal of Medical and Biological Research = Revista Brasileira De Pesquisas Medicas E Biologicas|April 14, 2016
Genetic aberrations in multiple myeloma characterized by cIg-FISH: a Brazilian contextP Segges, E Braggio, C Minnicelli, et al.
Journal of Clinical Pathology|May 29, 2004
Identification of three novel RB1 mutations in Brazilian patients with retinoblastoma by "exon by exon" PCR mediated SSCP analysisE Braggio, C R Bonvicino, F R Vargas, et al.
Blood Cancer Journal|September 23, 2017
High somatic mutation and neoantigen burden are correlated with decreased progression-free survival in multiple myelomaA Miller, Y Asmann, L Cattaneo, et al.
Leukemia|July 19, 2014
Monoclonal B-cell lymphocytosis is characterized by mutations in CLL putative driver genes and clonal heterogeneity many years before disease progressionJ Ojha, C Secreto, K Rabe, et al.
Leukemia|July 30, 2016
Genomic characterization of high-count MBL cases indicates that early detection of driver mutations and subclonal expansion are predictors of adverse clinical outcomeS Barrio, T D Shanafelt, J Ojha, et al.
Leukemia|January 21, 2012
Longitudinal genome-wide analysis of patients with chronic lymphocytic leukemia reveals complex evolution of clonal architecture at disease progression and at the time of relapseE Braggio, N E Kay, S VanWier, et al.
Blood Cancer Journal|February 27, 2016
Panel sequencing for clinically oriented variant screening and copy number detection in 142 untreated multiple myeloma patientsK M Kortuem, E Braggio, L Bruins, et al.
Leukemia|June 12, 2013
Genome-wide studies in multiple myeloma identify XPO1/CRM1 as a critical target validated using the selective nuclear export inhibitor KPT-276J Schmidt, E Braggio, K M Kortuem, et al.
Pageof 2