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Circulation
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May 29, 1998
Medical and cellular implications of stunning, hibernation, and preconditioning: an NHLBI workshop
R A Kloner, R Bolli, E Marban, et al.
Circulation
|
December 1, 1989
Calcium and its role in myocardial cell injury during ischemia and reperfusion
E Marban, Y Koretsune, M Corretti, et al.
The Journal of Physiology
|
November 1, 1991
Sodium channel inactivation from resting states in guinea-pig ventricular myocytes
J H Lawrence, D T Yue, W C Rose, et al.
Nature
|
August 28, 1980
Free calcium in heart muscle at rest and during contraction measured with Ca2+ -sensitive microelectrodes
E Marban, T J Rink, R W Tsien, et al.
The Journal of Physiology
|
October 1, 1992
Macroscopic and unitary properties of physiological ion flux through T-type Ca2+ channels in guinea-pig heart cells
C W Balke, W C Rose, E Marban, et al.
The Journal of Membrane Biology
|
March 1, 1997
Mechanism of Ca(2+)-dependent inactivation of L-type Ca2+ channels in GH3 cells: direct evidence against dephosphorylation by calcineurin
R G Victor, F Rusnak, R Sikkink, et al.
Circulation Research
|
March 1, 1994
Myofilament Ca2+ sensitivity in intact versus skinned rat ventricular muscle
W D Gao, P H Backx, M Azan-Backx, et al.
Advances in Experimental Medicine and Biology
|
January 1, 1993
Regulation of intracellular calcium in cardiac muscle
P H Backx, W D Gao, M D Azan-Backx, et al.
Biophysical Journal
|
April 17, 1998
Mechanism of anode break stimulation in the heart
R Ranjan, N Chiamvimonvat, N V Thakor, et al.
Circulation Research
|
May 1, 1990
Excitation-contraction coupling in postischemic myocardium. Does failure of activator Ca2+ transients underlie stunning?
H Kusuoka, Y Koretsune, V P Chacko, et al.
Page
of 14
Search research articles
Search
Showing results (81-90 of 138) with videos related to
Sort By:
Page
of 14
Circulation
|
May 29, 1998
Medical and cellular implications of stunning, hibernation, and preconditioning: an NHLBI workshop
R A Kloner, R Bolli, E Marban, et al.
Circulation
|
December 1, 1989
Calcium and its role in myocardial cell injury during ischemia and reperfusion
E Marban, Y Koretsune, M Corretti, et al.
The Journal of Physiology
|
November 1, 1991
Sodium channel inactivation from resting states in guinea-pig ventricular myocytes
J H Lawrence, D T Yue, W C Rose, et al.
Nature
|
August 28, 1980
Free calcium in heart muscle at rest and during contraction measured with Ca2+ -sensitive microelectrodes
E Marban, T J Rink, R W Tsien, et al.
The Journal of Physiology
|
October 1, 1992
Macroscopic and unitary properties of physiological ion flux through T-type Ca2+ channels in guinea-pig heart cells
C W Balke, W C Rose, E Marban, et al.
The Journal of Membrane Biology
|
March 1, 1997
Mechanism of Ca(2+)-dependent inactivation of L-type Ca2+ channels in GH3 cells: direct evidence against dephosphorylation by calcineurin
R G Victor, F Rusnak, R Sikkink, et al.
Circulation Research
|
March 1, 1994
Myofilament Ca2+ sensitivity in intact versus skinned rat ventricular muscle
W D Gao, P H Backx, M Azan-Backx, et al.
Advances in Experimental Medicine and Biology
|
January 1, 1993
Regulation of intracellular calcium in cardiac muscle
P H Backx, W D Gao, M D Azan-Backx, et al.
Biophysical Journal
|
April 17, 1998
Mechanism of anode break stimulation in the heart
R Ranjan, N Chiamvimonvat, N V Thakor, et al.
Circulation Research
|
May 1, 1990
Excitation-contraction coupling in postischemic myocardium. Does failure of activator Ca2+ transients underlie stunning?
H Kusuoka, Y Koretsune, V P Chacko, et al.
Page
of 14