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Physiological Genomics|July 23, 2015
AZT-induced mitochondrial toxicity: an epigenetic paradigm for dysregulation of gene expression through mitochondrial oxidative stressChristopher A Koczor, Zhe Jiao, Earl Fields, et al.
The American Journal of Pathology|April 4, 2012
p53 and mitochondrial DNA: their role in mitochondrial homeostasis and toxicity of antiretroviralsChristopher A Koczor, Richard C White, Peter Zhao, et al.
Physiological Genomics|January 14, 2016
Mitochondrial polymerase gamma dysfunction and aging cause cardiac nuclear DNA methylation changesChristopher A Koczor, Ivan Ludlow, Earl Fields, et al.
Ejhaem|February 26, 2024
Voxelotor improves red blood cell functionality in children with sickle cell anaemia: An ancillary study of the HOPE-KIDS 1 trialSatheesh Chonat, Earl Fields, Hannah Baratz, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|October 24, 2012
Transgenic mouse model with deficient mitochondrial polymerase exhibits reduced state IV respiration and enhanced cardiac fibrosisChristopher A Koczor, Rebecca A Torres, Earl Fields, et al.
Cardiovascular Toxicology|April 10, 2010
Absence of mitochondrial toxicity in hearts of transgenic mice treated with abacavirJames J Kohler, Seyed H Hosseini, Elgin Green, et al.
Toxicology and Applied Pharmacology|August 27, 2015
Methamphetamine and HIV-Tat alter murine cardiac DNA methylation and gene expressionChristopher A Koczor, Earl Fields, Mark J Jedrzejczak, et al.
British Journal of Haematology|July 20, 2025
Characterizing pregnancy outcomes in a humanized mouse model of sickle cell diseaseChristopher Chambliss, Elizabeth Manci, Earl Fields, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|August 8, 2015
Ecstasy (MDMA) Alters Cardiac Gene Expression and DNA Methylation: Implications for Circadian Rhythm Dysfunction in the HeartChristopher A Koczor, Ivan Ludlow, Robert S Hight, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 29, 2009
Transgenic mitochondrial superoxide dismutase and mitochondrially targeted catalase prevent antiretroviral-induced oxidative stress and cardiomyopathyJames J Kohler, Ioan Cucoranu, Earl Fields, et al.
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