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Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|October 20, 2007
DNA replication licensing factors and aurora kinases are linked to aneuploidy and clinical outcome in epithelial ovarian carcinomaAnjana A Kulkarni, Marco Loddo, Elisabetta Leo, et al.
Cancers|August 26, 2023
Relevance of ATM Status in Driving Sensitivity to DNA Damage Response Inhibitors in Patient-Derived Xenograft ModelsAnkur Karmokar, Rebecca Sargeant, Adina M Hughes, et al.
Molecular Cell|February 4, 2018
SLFN11 Blocks Stressed Replication Forks Independently of ATRJunko Murai, Sai-Wen Tang, Elisabetta Leo, et al.
JCI Insight|September 22, 2021
SLFN11 captures cancer-immunity interactions associated with platinum sensitivity in high-grade serous ovarian cancerClaudia Winkler, Matthew King, Julie Berthe, et al.
Oncotarget|May 14, 2019
Rapid activation of epithelial-mesenchymal transition drives PARP inhibitor resistance in Brca2-mutant mammary tumoursLiliana D Ordonez, Trevor Hay, Robert McEwen, et al.
NAR Cancer|October 9, 2025
Androgen receptor inhibition extends PARP inhibitor activity in prostate cancer models beyond BRCA mutations and defects in homologous recombination repairGiuditta Illuzzi, Alessandro Galbiati, Anna D Staniszewska, et al.
Oncoimmunology|June 27, 2022
PARP inhibition is a modulator of anti-tumor immune response in BRCA-deficient tumorsAnna D Staniszewska, Joshua Armenia, Matthew King, et al.
The Biochemical Journal|December 9, 2014
Observed bromodomain flexibility reveals histone peptide- and small molecule ligand-compatible forms of ATAD2Guillaume Poncet-Montange, Yanai Zhan, Jennifer P Bardenhagen, et al.
British Journal of Cancer|December 19, 2020
SLFN11 informs on standard of care and novel treatments in a wide range of cancer modelsClaudia Winkler, Joshua Armenia, Gemma N Jones, et al.
The EMBO Journal|August 17, 2010
Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replicationDongyi Xu, Parameswary Muniandy, Elisabetta Leo, et al.
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