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Journal of the American Heart Association|November 7, 2012
Measurement of reverse cholesterol transport pathways in humans: in vivo rates of free cholesterol efflux, esterification, and excretionScott Turner, Jason Voogt, Michael Davidson, et al.
Circulation Research|April 30, 2016
Mitochondrial Function, Biology, and Role in Disease: A Scientific Statement From the American Heart AssociationElizabeth Murphy, Hossein Ardehali, Robert S Balaban, et al.
International Journal of Prisoner Health|November 24, 2018
A novel care pathway for prisoners with intellectual disability designed through a Delphi processGautam Gulati, Stephen Quigley, Valerie Elizabeth Murphy, et al.
Journal of Molecular and Cellular Cardiology|June 10, 2015
Assessment of cardiac function in mice lacking the mitochondrial calcium uniporterKira M Holmström, Xin Pan, Julia C Liu, et al.
Scientific Reports|May 20, 2017
Parkin regulation of CHOP modulates susceptibility to cardiac endoplasmic reticulum stressKim Han, Shahin Hassanzadeh, Komudi Singh, et al.
Cell Reports|August 2, 2016
MICU1 Serves as a Molecular Gatekeeper to Prevent In Vivo Mitochondrial Calcium OverloadJulia C Liu, Jie Liu, Kira M Holmström, et al.
Nature Communications|April 14, 2026
Loss of TMEM65 in mice causes mitochondrial disease mediated by mitochondrial Ca2Yingfan Zhang, Hailey A Parry, Laura Reyes, et al.
Circulation Research|February 28, 2004
Nuclear targeting of Akt enhances kinase activity and survival of cardiomyocytesIsao Shiraishi, Jaime Melendez, Youngkeun Ahn, et al.
The Journal of Clinical Investigation|October 5, 2010
Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in miceJohn W Elrod, Renee Wong, Shikha Mishra, et al.
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