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Experimental Gerontology|April 5, 2014
The paradox of mitochondrial dysfunction and extended longevityErin Munkácsy, Shane L ReaAging|October 11, 2013
TAF-4 is required for the life extension of isp-1, clk-1 and tpk-1 Mit mutantsMaruf H Khan, Melissa Ligon, Lauren R Hussey, et al.Aging Cell|January 6, 2016
Mitochondrial metabolites extend lifespanRobert J Mishur, Maruf Khan, Erin Munkácsy, et al.Experimental Gerontology|September 3, 2005
Metabolism in the Caenorhabditis elegans Mit mutantsShane L ReaPlos Genetics|July 16, 2016
DLK-1, SEK-3 and PMK-3 Are Required for the Life Extension Induced by Mitochondrial Bioenergetic Disruption in C. elegansErin Munkácsy, Maruf H Khan, Rebecca K Lane, et al.Biotechnology Journal|April 20, 2007
Caenorhabditis elegans mitochondrial mutants as an investigative tool to study human neurodegenerative diseases associated with mitochondrial dysfunctionNatascia Ventura, Shane L ReaMass Spectrometry Reviews|May 4, 2011
Applications of mass spectrometry to metabolomics and metabonomics: detection of biomarkers of aging and of age-related diseasesRobert J Mishur, Shane L ReaBiorxiv : the Preprint Server for Biology|December 9, 2024
Deep Learning of Cellular Metabolic Flux Distributions Predicts LifespanTyler A U Hilsabeck, Shane L ReaExperimental Gerontology|September 2, 2006
Long-lived C. elegans mitochondrial mutants as a model for human mitochondrial-associated diseasesNatascia Ventura, Shane L Rea, Roberto TestiPageof 4