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Biotechniques|September 21, 2000
Combining enhanced metabolic labeling with immunoblotting to detect interactions of endogenous cellular proteinsF C Kischkel, A AshkenaziImmunity|July 14, 2000
Apo2L/TRAIL-dependent recruitment of endogenous FADD and caspase-8 to death receptors 4 and 5F C Kischkel, D A Lawrence, A Chuntharapai, et al.The EMBO Journal|November 15, 1995
Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptorF C Kischkel, S Hellbardt, I Behrmann, et al.Cell Death and Differentiation|April 1, 1996
CD95 (APO-1/Fas)-associating signalling proteinsM E Peter, F C Kischkel, S Hellbardt, et al.The EMBO Journal|May 15, 1997
FLICE is activated by association with the CD95 death-inducing signaling complex (DISC)J P Medema, C Scaffidi, F C Kischkel, et al.European Journal of Immunology|May 1, 1997
Resistance of cultured peripheral T cells towards activation-induced cell death involves a lack of recruitment of FLICE (MACH/caspase 8) to the CD95 death-inducing signaling complexM E Peter, F C Kischkel, C G Scheuerpflug, et al.Oncogene|November 21, 1996
CD95 (APO-1/Fas) induces activation of SAP kinases downstream of ICE-like proteasesM A Cahill, M E Peter, F C Kischkel, et al.The Journal of Biological Chemistry|October 5, 2001
Death receptor recruitment of endogenous caspase-10 and apoptosis initiation in the absence of caspase-8F C Kischkel, D A Lawrence, A Tinel, et al.The Journal of Biological Chemistry|March 1, 1996
FADD/MORT1 is a common mediator of CD95 (Fas/APO-1) and tumor necrosis factor receptor-induced apoptosisA M Chinnaiyan, C G Tepper, M F Seldin, et al.Cell|June 14, 1996
FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complexM Muzio, A M Chinnaiyan, F C Kischkel, et al.Pageof 2