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Cancer Research|December 11, 1997
Absence of Fhit protein in primary lung tumors and cell lines with FHIT gene abnormalitiesG Sozzi, S Tornielli, E Tagliabue, et al.Proceedings of the National Academy of Sciences of the United States of America|July 22, 1998
Nitrilase and Fhit homologs are encoded as fusion proteins in Drosophila melanogaster and Caenorhabditis elegansY Pekarsky, M Campiglio, Z Siprashvili, et al.Proceedings of the National Academy of Sciences of the United States of America|February 12, 1998
Replacement of Fhit in cancer cells suppresses tumorigenicityZ Siprashvili, G Sozzi, L D Barnes, et al.Proceedings of the National Academy of Sciences of the United States of America|April 12, 2000
Muir-Torre-like syndrome in Fhit-deficient miceL Y Fong, V Fidanza, N Zanesi, et al.Cell Death and Differentiation|September 5, 2009
miR-145 participates with TP53 in a death-promoting regulatory loop and targets estrogen receptor-alpha in human breast cancer cellsR Spizzo, M S Nicoloso, L Lupini, et al.Molecular and Cellular Biology|March 22, 2001
Critical role of the HMGI(Y) proteins in adipocytic cell growth and differentiationR M Melillo, G M Pierantoni, S Scala, et al.Cell|April 5, 1996
The FHIT gene 3p14.2 is abnormal in lung cancerG Sozzi, M L Veronese, M Negrini, et al.Scientific Reports|January 18, 2020
Endogenous and artificial miRNAs explore a rich variety of conformations: a potential relationship between secondary structure and biological functionalityC M A Gangemi, S Alaimo, A Pulvirenti, et al.Cancer Journal (Sudbury, Mass.)|July 6, 2000
Adjuvant high-dose interferon alfa-2b in patients with high-risk melanomaE Jonasch, U N Kumar, G P Linette, et al.Proceedings of the National Academy of Sciences of the United States of America|August 9, 2001
Heart-targeted overexpression of caspase3 in mice increases infarct size and depresses cardiac functionG Condorelli, R Roncarati, J Ross, et al.Pageof 52