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Feng-Chun Yang

Showing results (91-100 of 126) with videos related to

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Stem Cell Reports|May 31, 2016
Loss of Asxl1 Alters Self-Renewal and Cell Fate of Bone Marrow Stromal Cell, Leading to Bohring-Opitz-like Syndrome in MicePeng Zhang, Caihong Xing, Steven D Rhodes, et al.
Genomics, Proteomics & Bioinformatics|June 17, 2018
Tet2 Regulates Osteoclast Differentiation by Interacting with Runx1 and Maintaining Genomic 5-Hydroxymethylcytosine (5hmC)Yajing Chu, Zhigang Zhao, David Wayne Sant, et al.
Cell Reports|November 21, 2015
Combined Loss of Tet1 and Tet2 Promotes B Cell, but Not Myeloid Malignancies, in MiceZhigang Zhao, Li Chen, Meelad M Dawlaty, et al.
The Journal of Cell Biology|June 19, 2013
The tumor suppressor CDKN3 controls mitosisGrzegorz Nalepa, Jill Barnholtz-Sloan, Rikki Enzor, et al.
Blood|April 7, 2017
Caspase-3 controls AML1-ETO-driven leukemogenesis via autophagy modulation in a ULK1-dependent mannerNa Man, Yurong Tan, Xiao-Jian Sun, et al.
The Journal of Clinical Investigation|December 11, 2012
Normal hematopoiesis and neurofibromin-deficient myeloproliferative disease require ErkKarl Staser, Su-Jung Park, Steven D Rhodes, et al.
Nature Communications|December 31, 2024
DDX18 coordinates nucleolus phase separation and nuclear organization to control the pluripotency of human embryonic stem cellsXianle Shi, Yanjing Li, Hongwei Zhou, et al.
Cell|November 6, 2008
Nf1-dependent tumors require a microenvironment containing Nf1+/-- and c-kit-dependent bone marrowFeng-Chun Yang, David A Ingram, Shi Chen, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 25, 2013
Hyperactive transforming growth factor-β1 signaling potentiates skeletal defects in a neurofibromatosis type 1 mouse modelSteven D Rhodes, Xiaohua Wu, Yongzheng He, et al.
The Journal of Clinical Investigation|November 2, 2023
Targeting lysine demethylase 6B ameliorates ASXL1 truncation-mediated myeloid malignancies in preclinical modelsGuo Ge, Peng Zhang, Pinpin Sui, et al.
Pageof 13

Showing results (91-100 of 126) with videos related to

Sort By:
Pageof 13
Stem Cell Reports|May 31, 2016
Loss of Asxl1 Alters Self-Renewal and Cell Fate of Bone Marrow Stromal Cell, Leading to Bohring-Opitz-like Syndrome in MicePeng Zhang, Caihong Xing, Steven D Rhodes, et al.
Genomics, Proteomics & Bioinformatics|June 17, 2018
Tet2 Regulates Osteoclast Differentiation by Interacting with Runx1 and Maintaining Genomic 5-Hydroxymethylcytosine (5hmC)Yajing Chu, Zhigang Zhao, David Wayne Sant, et al.
Cell Reports|November 21, 2015
Combined Loss of Tet1 and Tet2 Promotes B Cell, but Not Myeloid Malignancies, in MiceZhigang Zhao, Li Chen, Meelad M Dawlaty, et al.
The Journal of Cell Biology|June 19, 2013
The tumor suppressor CDKN3 controls mitosisGrzegorz Nalepa, Jill Barnholtz-Sloan, Rikki Enzor, et al.
Blood|April 7, 2017
Caspase-3 controls AML1-ETO-driven leukemogenesis via autophagy modulation in a ULK1-dependent mannerNa Man, Yurong Tan, Xiao-Jian Sun, et al.
The Journal of Clinical Investigation|December 11, 2012
Normal hematopoiesis and neurofibromin-deficient myeloproliferative disease require ErkKarl Staser, Su-Jung Park, Steven D Rhodes, et al.
Nature Communications|December 31, 2024
DDX18 coordinates nucleolus phase separation and nuclear organization to control the pluripotency of human embryonic stem cellsXianle Shi, Yanjing Li, Hongwei Zhou, et al.
Cell|November 6, 2008
Nf1-dependent tumors require a microenvironment containing Nf1+/-- and c-kit-dependent bone marrowFeng-Chun Yang, David A Ingram, Shi Chen, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 25, 2013
Hyperactive transforming growth factor-β1 signaling potentiates skeletal defects in a neurofibromatosis type 1 mouse modelSteven D Rhodes, Xiaohua Wu, Yongzheng He, et al.
The Journal of Clinical Investigation|November 2, 2023
Targeting lysine demethylase 6B ameliorates ASXL1 truncation-mediated myeloid malignancies in preclinical modelsGuo Ge, Peng Zhang, Pinpin Sui, et al.
Pageof 13