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Oncogene|December 4, 2003
Constitutive DNA damage is linked to DNA replication abnormalities in Bloom's syndrome cellsFeyruz V Rassool, Philip S North, Ghulam J Mufti, et al.
Advances in Cancer Research|January 30, 2019
Exploiting epigenetically mediated changes: Acute myeloid leukemia, leukemia stem cells and the bone marrow microenvironmentAksinija A Kogan, Rena G Lapidus, Maria R Baer, et al.
Cancer Research|April 19, 2003
Increased error-prone NHEJ activity in myeloid leukemias is associated with DNA damage at sites that recruit key nonhomologous end-joining proteinsNicola Brady, Terry J Gaymes, Manyee Cheung, et al.
Leukemia Research|August 21, 2013
Oxidative stress leads to increased mutation frequency in a murine model of myelodysplastic syndromeYang Jo Chung, Carine Robert, Sheryl M Gough, et al.
Blood|January 14, 2011
Defective nonhomologous end joining blocks B-cell development in FLT3/ITD miceLi Li, Li Zhang, Jinshui Fan, et al.
Cancer Discovery|October 4, 2024
The Epigenetic Hallmarks of CancerManel Esteller, Mark A Dawson, Cigall Kadoch, et al.
Oncogene|April 24, 2002
Increased error-prone non homologous DNA end-joining--a proposed mechanism of chromosomal instability in Bloom's syndromeTerry J Gaymes, Phillip S North, Nicola Brady, et al.
Stem Cells International|October 1, 2016
High-Fidelity Reprogrammed Human IPSCs Have a High Efficacy of DNA Repair and Resemble hESCs in Their MYC Transcriptional SignaturePratik K Nagaria, Carine Robert, Tea Soon Park, et al.
Molecular Cancer Therapeutics|May 16, 2015
Aromatase Inhibitor-Mediated Downregulation of INrf2 (Keap1) Leads to Increased Nrf2 and Resistance in Breast CancerRaju Khatri, Preeti Shah, Rupa Guha, et al.
Translational Oncology|November 22, 2021
PARP1 PARylates and stabilizes STAT5 in FLT3-ITD acute myeloid leukemia and other STAT5-activated cancersAnna J Dellomo, Rachel Abbotts, Christian L Eberly, et al.
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