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Clinical and Vaccine Immunology : CVI
|
November 6, 2007
Human skin endothelial cells can express all 10 TLR genes and respond to respective ligands
Nicole Fitzner, Sigrid Clauberg, Frank Essmann, et al.
Journal of Experimental Therapeutics & Oncology
|
June 26, 2003
A rapid nonradioactive peptide phosphorylation assay
Martin Zeller, Frank Essmann, Reiner U Jänicke, et al.
Cancer Research
|
October 7, 2004
Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of caspase-3 and a caffeine-inhibitable event
Frank Essmann, Ingo H Engels, Gudrun Totzke, et al.
Assay and Drug Development Technologies
|
December 24, 2011
Kinetic tracking of therapy-induced senescence using the real-time cell analyzer single plate system
Timo Weiland, Alexander Berger, Frank Essmann, et al.
Cell Cycle (Georgetown, Tex.)
|
May 16, 2013
α-Fucosidase as a novel convenient biomarker for cellular senescence
Dominic G Hildebrand, Simon Lehle, Andreas Borst, et al.
Oncotarget
|
January 20, 2018
Simultaneous quantification of DNA damage and mitochondrial copy number by long-run DNA-damage quantification (LORD-Q)
Benjamin Dannenmann, Simon Lehle, Sebastian Lorscheid, et al.
The Journal of Biological Chemistry
|
March 4, 2006
A novel member of the IkappaB family, human IkappaB-zeta, inhibits transactivation of p65 and its DNA binding
Gudrun Totzke, Frank Essmann, Stephan Pohlmann, et al.
Molecular and Cellular Biology
|
February 16, 2006
The proteasome is required for rapid initiation of death receptor-induced apoptosis
Dennis Sohn, Gudrun Totzke, Frank Essmann, et al.
Natural Product Research
|
July 18, 2015
A new quinolinone and its natural/artificial derivatives from a shark gill-derived fungus Penicillium polonicum AP2T1
Yi Zhang, Jun Mu, Frank Essmann, et al.
Oncogene
|
April 18, 2003
Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop
Clarissa von Haefen, Thomas Wieder, Frank Essmann, et al.
Page
of 7
Search research articles
Search
Showing results (11-20 of 61) with videos related to
Sort By:
Page
of 7
Clinical and Vaccine Immunology : CVI
|
November 6, 2007
Human skin endothelial cells can express all 10 TLR genes and respond to respective ligands
Nicole Fitzner, Sigrid Clauberg, Frank Essmann, et al.
Journal of Experimental Therapeutics & Oncology
|
June 26, 2003
A rapid nonradioactive peptide phosphorylation assay
Martin Zeller, Frank Essmann, Reiner U Jänicke, et al.
Cancer Research
|
October 7, 2004
Apoptosis resistance of MCF-7 breast carcinoma cells to ionizing radiation is independent of p53 and cell cycle control but caused by the lack of caspase-3 and a caffeine-inhibitable event
Frank Essmann, Ingo H Engels, Gudrun Totzke, et al.
Assay and Drug Development Technologies
|
December 24, 2011
Kinetic tracking of therapy-induced senescence using the real-time cell analyzer single plate system
Timo Weiland, Alexander Berger, Frank Essmann, et al.
Cell Cycle (Georgetown, Tex.)
|
May 16, 2013
α-Fucosidase as a novel convenient biomarker for cellular senescence
Dominic G Hildebrand, Simon Lehle, Andreas Borst, et al.
Oncotarget
|
January 20, 2018
Simultaneous quantification of DNA damage and mitochondrial copy number by long-run DNA-damage quantification (LORD-Q)
Benjamin Dannenmann, Simon Lehle, Sebastian Lorscheid, et al.
The Journal of Biological Chemistry
|
March 4, 2006
A novel member of the IkappaB family, human IkappaB-zeta, inhibits transactivation of p65 and its DNA binding
Gudrun Totzke, Frank Essmann, Stephan Pohlmann, et al.
Molecular and Cellular Biology
|
February 16, 2006
The proteasome is required for rapid initiation of death receptor-induced apoptosis
Dennis Sohn, Gudrun Totzke, Frank Essmann, et al.
Natural Product Research
|
July 18, 2015
A new quinolinone and its natural/artificial derivatives from a shark gill-derived fungus Penicillium polonicum AP2T1
Yi Zhang, Jun Mu, Frank Essmann, et al.
Oncogene
|
April 18, 2003
Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop
Clarissa von Haefen, Thomas Wieder, Frank Essmann, et al.
Page
of 7