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G D Zeevalk

Showing results (11-20 of 44) with videos related to

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Neuroscience Letters|March 14, 1986
Glutamate and kainate are not directly toxic to developing amacrine cells: analysis in a Lucifer Yellow-labeled populationG D Zeevalk, A G Hyndman
Brain Research|December 15, 1987
Transferrin in chick retina: distribution and location during developmentG D Zeevalk, A G Hyndman
Experimental Eye Research|September 1, 1997
Contribution of glial metabolism to neuronal damage caused by partial inhibition of energy metabolism in retinaG D Zeevalk, W J Nicklas
Brain Research|July 2, 1990
Action of the anti-ischemic agent ifenprodil on N-methyl-D-aspartate and kainate-mediated excitotoxicityG D Zeevalk, W J Nicklas
Brain Research. Developmental Brain Research|February 21, 1992
Developmental differences in antagonism of NMDA toxicity by the polyamine site antagonist ifenprodilG D Zeevalk, W J Nicklas
Neuroscience Letters|July 31, 1989
Acute excitotoxicity in chick retina caused by the unusual amino acids BOAA and BMAA: effects of MK-801 and kynurenateG D Zeevalk, W J Nicklas
Journal of Neurochemistry|August 11, 2000
Lactate prevents the alterations in tissue amino acids, decline in ATP, and cell damage due to aglycemia in retinaG D Zeevalk, W J Nicklas
The Journal of Pharmacology and Experimental Therapeutics|October 1, 1996
Hypothermia and metabolic stress: narrowing the cellular site of early neuroprotectionG D Zeevalk, W J Nicklas
Molecular and Chemical Neuropathology|September 1, 1996
Attenuation of excitotoxic cell swelling and GABA release by the GABA transport inhibitor SKF 89976AG D Zeevalk, W J Nicklas
Journal of Neurochemistry|July 1, 1993
Aurintricarboxylic acid prevents NMDA-mediated excitotoxicity: evidence for its action as an NMDA receptor antagonistG D Zeevalk, D Schoepp, W J Nicklas
Pageof 5

Showing results (11-20 of 44) with videos related to

Sort By:
Pageof 5
Neuroscience Letters|March 14, 1986
Glutamate and kainate are not directly toxic to developing amacrine cells: analysis in a Lucifer Yellow-labeled populationG D Zeevalk, A G Hyndman
Brain Research|December 15, 1987
Transferrin in chick retina: distribution and location during developmentG D Zeevalk, A G Hyndman
Experimental Eye Research|September 1, 1997
Contribution of glial metabolism to neuronal damage caused by partial inhibition of energy metabolism in retinaG D Zeevalk, W J Nicklas
Brain Research|July 2, 1990
Action of the anti-ischemic agent ifenprodil on N-methyl-D-aspartate and kainate-mediated excitotoxicityG D Zeevalk, W J Nicklas
Brain Research. Developmental Brain Research|February 21, 1992
Developmental differences in antagonism of NMDA toxicity by the polyamine site antagonist ifenprodilG D Zeevalk, W J Nicklas
Neuroscience Letters|July 31, 1989
Acute excitotoxicity in chick retina caused by the unusual amino acids BOAA and BMAA: effects of MK-801 and kynurenateG D Zeevalk, W J Nicklas
Journal of Neurochemistry|August 11, 2000
Lactate prevents the alterations in tissue amino acids, decline in ATP, and cell damage due to aglycemia in retinaG D Zeevalk, W J Nicklas
The Journal of Pharmacology and Experimental Therapeutics|October 1, 1996
Hypothermia and metabolic stress: narrowing the cellular site of early neuroprotectionG D Zeevalk, W J Nicklas
Molecular and Chemical Neuropathology|September 1, 1996
Attenuation of excitotoxic cell swelling and GABA release by the GABA transport inhibitor SKF 89976AG D Zeevalk, W J Nicklas
Journal of Neurochemistry|July 1, 1993
Aurintricarboxylic acid prevents NMDA-mediated excitotoxicity: evidence for its action as an NMDA receptor antagonistG D Zeevalk, D Schoepp, W J Nicklas
Pageof 5