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The Cochrane Database of Systematic Reviews|October 21, 2004
Immunosuppressive treatment for idiopathic membranous nephropathy in adults with nephrotic syndromeA Schieppati, A Perna, J Zamora, et al.Journal of the American Society of Nephrology : JASN|December 1, 1994
Effects of angiotensin-converting enzyme inhibition on glomerular capillary wall ultrastructure in MWF/Ztm ratsB E Iordache, O Imberti, C Foglieni, et al.Journal of the American Society of Nephrology : JASN|October 17, 1998
Chronic allograft nephropathy in the rat is improved by angiotensin II receptor blockade but not by calcium channel antagonismS C Amuchastegui, N Azzollini, M Mister, et al.Lancet (London, England)|December 10, 1977
Prostacyclin-like activity and bleeding in renal failureG Remuzzi, A E Cavenaghi, G Mecca, et al.Nephron|August 15, 2000
Nonviral and viral gene transfer to the kidney in the context of transplantationA Benigni, S Tomasoni, J Lutz, et al.The Journal of Laboratory and Clinical Medicine|September 1, 1988
Prolonged bleeding time and increased vascular prostacyclin in rats with chronic renal failure: effects of conjugated estrogensC Zoja, G Viganò, A Bergamelli, et al.American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|March 1, 1991
Low-protein diet and glomerular size-selective function in membranous glomerulopathyA Remuzzi, E Perticucci, C Battaglia, et al.The Journal of Pharmacology and Experimental Therapeutics|January 1, 1990
17 beta-estradiol is the most active component of the conjugated estrogen mixture active on uremic bleeding by a receptor mechanismG Viganò, C Zoja, D Corna, et al.The American Journal of Physiology|March 10, 1999
ACE inhibition and ANG II receptor blockade improve glomerular size-selectivity in IgA nephropathyA Remuzzi, N Perico, F Sangalli, et al.Kidney International|February 23, 1999
Glomerular size-selective dysfunction in NIDDM is not ameliorated by ACE inhibition or by calcium channel blockadeP Ruggenenti, L Mosconi, F Sangalli, et al.Pageof 41