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Archives D'Anatomie Et De Cytologie Pathologiques|October 1, 1998
[Origin and progression of thyroid epithelial tumors: molecular and cellular mechanisms]V Gire, D Wynford-ThomasOncogene|March 4, 2000
RAS oncogene activation induces proliferation in normal human thyroid epithelial cells without loss of differentiationV Gire, D Wynford-ThomasBritish Journal of Cancer|June 1, 1995
Does autocrine growth factor secretion form part of a mechanism which paradoxically protects against tumour development?T Dawson, D Wynford-ThomasExperimental Cell Research|September 1, 1986
Heterogeneity in distribution and content of p53 in SV40-transformed mouse fibroblastsD Davis, D Wynford-ThomasCarcinogenesis|February 24, 1998
The influence of cell context on the selection pressure for p53 mutation in human cancerD Wynford-Thomas, J BlaydesBritish Journal of Cancer|February 1, 1988
Ha-ras restriction fragment length polymorphisms in colorectal cancerF S Wyllie, V Wynford-Thomas, N R Lemoine, et al.Oncogene|July 29, 1998
The proliferation of normal human fibroblasts is dependent upon negative regulation of p53 function by mdm2J P Blaydes, D Wynford-ThomasThe Journal of Pathology|October 1, 1990
The polymerase chain reaction: miracle or mirage? A critical review of its uses and limitations in diagnosis and researchP A Wright, D Wynford-ThomasInternational Journal of Cancer|February 8, 1996
Loss of responsiveness to transforming growth factor beta (TGFbeta) is tightly linked to tumorigenicity in a model of thyroid tumour progressionJ P Blaydes, D Wynford-ThomasActa Endocrinologica|November 1, 1982
Mast cell hyperplasia during goitrogen-induced thyroid growth - a quantitative studyD Wynford-Thomas, B M StringerPageof 34