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G Todd Milne

Showing results (1-10 of 15) with videos related to

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Molecular and Cellular Biology|October 16, 2004
Identification of the proteins required for biosynthesis of diphthamide, the target of bacterial ADP-ribosylating toxins on translation elongation factor 2Shihui Liu, G Todd Milne, Jeffrey G Kuremsky, et al.
Handbook of Experimental Pharmacology|July 6, 2019
Correction to: Soluble Guanylate Cyclase Stimulators and ActivatorsPeter Sandner, Daniel P Zimmer, G Todd Milne, et al.
Handbook of Experimental Pharmacology|January 29, 2019
Soluble Guanylate Cyclase Stimulators and ActivatorsPeter Sandner, Daniel P Zimmer, G Todd Milne, et al.
Plos One|May 3, 2014
Fatty acid amide hydrolase (FAAH) inhibitors exert pharmacological effects, but lack antinociceptive efficacy in rats with neuropathic spinal cord injury painAldric T Hama, Peter Germano, Matthew S Varghese, et al.
Cellular and Molecular Life Sciences : CMLS|October 14, 2024
Pharmacologically increasing cGMP improves proteostasis and reduces neuropathy in mouse models of CMT1Seth M Moore, Joseph Gawron, Mckayla Stevens, et al.
JACC. Basic to Translational Science|September 18, 2023
Stimulation of Erythrocyte Soluble Guanylyl Cyclase Induces cGMP Export and Cardioprotection in Type 2 DiabetesTong Jiao, Aida Collado, Ali Mahdi, et al.
Antimicrobial Agents and Chemotherapy|September 29, 2006
Inhibition of filamentation can be used to treat disseminated candidiasisStephen P Saville, Anna L Lazzell, Alexander P Bryant, et al.
The Journal of Pharmacology and Experimental Therapeutics|October 23, 2010
Fatty acid amide hydrolase (FAAH) inhibition reduces L-3,4-dihydroxyphenylalanine-induced hyperactivity in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned non-human primate model of Parkinson's diseaseTom H Johnston, Philippe Huot, Susan H Fox, et al.
Clinical Pharmacology in Drug Development|November 14, 2018
A Randomized, Placebo-Controlled, Multiple-Ascending-Dose Study to Assess the Safety, Tolerability, Pharmacokinetics, and Pharmacodynamics of the Soluble Guanylate Cyclase Stimulator Praliciguat in Healthy SubjectsJohn P Hanrahan, James D Wakefield, Phebe J Wilson, et al.
Pharmacology Research & Perspectives|April 22, 2020
Pharmacokinetics, mass balance, tissue distribution, metabolism, and excretion of praliciguat, a clinical-stage soluble guanylate cyclase stimulator in ratsAli R Banijamali, Andrew E Carvalho, James D Wakefield, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Molecular and Cellular Biology|October 16, 2004
Identification of the proteins required for biosynthesis of diphthamide, the target of bacterial ADP-ribosylating toxins on translation elongation factor 2Shihui Liu, G Todd Milne, Jeffrey G Kuremsky, et al.
Handbook of Experimental Pharmacology|July 6, 2019
Correction to: Soluble Guanylate Cyclase Stimulators and ActivatorsPeter Sandner, Daniel P Zimmer, G Todd Milne, et al.
Handbook of Experimental Pharmacology|January 29, 2019
Soluble Guanylate Cyclase Stimulators and ActivatorsPeter Sandner, Daniel P Zimmer, G Todd Milne, et al.
Plos One|May 3, 2014
Fatty acid amide hydrolase (FAAH) inhibitors exert pharmacological effects, but lack antinociceptive efficacy in rats with neuropathic spinal cord injury painAldric T Hama, Peter Germano, Matthew S Varghese, et al.
Cellular and Molecular Life Sciences : CMLS|October 14, 2024
Pharmacologically increasing cGMP improves proteostasis and reduces neuropathy in mouse models of CMT1Seth M Moore, Joseph Gawron, Mckayla Stevens, et al.
JACC. Basic to Translational Science|September 18, 2023
Stimulation of Erythrocyte Soluble Guanylyl Cyclase Induces cGMP Export and Cardioprotection in Type 2 DiabetesTong Jiao, Aida Collado, Ali Mahdi, et al.
Antimicrobial Agents and Chemotherapy|September 29, 2006
Inhibition of filamentation can be used to treat disseminated candidiasisStephen P Saville, Anna L Lazzell, Alexander P Bryant, et al.
The Journal of Pharmacology and Experimental Therapeutics|October 23, 2010
Fatty acid amide hydrolase (FAAH) inhibition reduces L-3,4-dihydroxyphenylalanine-induced hyperactivity in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned non-human primate model of Parkinson's diseaseTom H Johnston, Philippe Huot, Susan H Fox, et al.
Clinical Pharmacology in Drug Development|November 14, 2018
A Randomized, Placebo-Controlled, Multiple-Ascending-Dose Study to Assess the Safety, Tolerability, Pharmacokinetics, and Pharmacodynamics of the Soluble Guanylate Cyclase Stimulator Praliciguat in Healthy SubjectsJohn P Hanrahan, James D Wakefield, Phebe J Wilson, et al.
Pharmacology Research & Perspectives|April 22, 2020
Pharmacokinetics, mass balance, tissue distribution, metabolism, and excretion of praliciguat, a clinical-stage soluble guanylate cyclase stimulator in ratsAli R Banijamali, Andrew E Carvalho, James D Wakefield, et al.
Pageof 2