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Methods in Molecular Biology (Clifton, N.J.)|June 18, 2004
Mouse models of triplet repeat diseasesGillian P Bates, David G HayNature Reviews. Genetics|September 2, 2005
History of genetic disease: the molecular genetics of Huntington disease - a historyGillian P BatesNeuron|February 6, 2025
Somatic CAG-repeat expansion drives neuronal loss in Huntington's diseaseGillian P BatesNature Reviews. Neuroscience|September 22, 2006
Histone deacetylase inhibitors as therapeutics for polyglutamine disordersRachel Butler, Gillian P BatesAdvances in Experimental Medicine and Biology|February 11, 2018
RNA Related Pathology in Huntington's DiseaseAndreas Neueder, Gillian P BatesCurrent Opinion in Neurology|July 19, 2003
Experimental therapeutics in Huntington's disease: are models useful for therapeutic trials?Gillian P Bates, Emma HocklyEMBO Reports|October 2, 2004
Huntingtin and the molecular pathogenesis of Huntington's disease. Fourth in molecular medicine review seriesChristian Landles, Gillian P BatesMolecular Biotechnology|January 31, 2006
Mouse models of triplet repeat diseasesGillian P Bates, Roman GonitelBMC Medical Genomics|November 1, 2014
A common gene expression signature in Huntington's disease patient brain regionsAndreas Neueder, Gillian P BatesHuman Molecular Genetics|April 30, 2004
Progressive decrease in chaperone protein levels in a mouse model of Huntington's disease and induction of stress proteins as a therapeutic approachDavid G Hay, Kirupa Sathasivam, Sönke Tobaben, et al.Pageof 15