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The Biochemical Journal|August 5, 2022
Cell death in skin function, inflammation, and diseaseHolly Anderton, Suhaib AlqudahNature Reviews. Rheumatology|July 10, 2020
Cell death in chronic inflammation: breaking the cycle to treat rheumatic diseaseHolly Anderton, Ian P Wicks, John SilkeThe Journal of Investigative Dermatology|June 26, 2017
Inhibitor of Apoptosis Proteins (IAPs) Limit RIPK1-Mediated Skin InflammationHolly Anderton, James A Rickard, George A Varigos, et al.Cell Death & Disease|March 27, 2023
Deletion of Gpatch2 does not alter Tnf expression in miceDestiny Dalseno, Holly Anderton, Andrew Kueh, et al.Cell Reports|June 8, 2022
Langerhans cells are an essential cellular intermediary in chronic dermatitisHolly Anderton, Michaël Chopin, Caleb A Dawson, et al.Immunology and Cell Biology|January 20, 2022
ZC3H12C expression in dendritic cells is necessary to prevent lymphadenopathy of skin-draining lymph nodesElise Clayer, Daniel Frank, Holly Anderton, et al.Blood|February 9, 2011
Deletion of cIAP1 and cIAP2 in murine B lymphocytes constitutively activates cell survival pathways and inactivates the germinal center responseSandra Gardam, Vivian M Turner, Holly Anderton, et al.EMBO Reports|December 15, 2010
Cyclic-AMP-dependent protein kinase A regulates apoptosis by stabilizing the BH3-only protein BimDiane Moujalled, Ross Weston, Holly Anderton, et al.The Journal of Biological Chemistry|April 2, 2010
Tumor necrosis factor (TNF) signaling, but not TWEAK (TNF-like weak inducer of apoptosis)-triggered cIAP1 (cellular inhibitor of apoptosis protein 1) degradation, requires cIAP1 RING dimerization and E2 bindingRebecca Feltham, Maryline Moulin, James E Vince, et al.The EMBO Journal|February 14, 2012
IAPs limit activation of RIP kinases by TNF receptor 1 during developmentMaryline Moulin, Holly Anderton, Anne K Voss, et al.Pageof 4