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Diabetes|July 1, 1996
K+ channels: generating excitement in pancreatic beta-cellsI D Dukes, L H PhilipsonThe Journal of Biological Chemistry|April 15, 1994
Dependence on NADH produced during glycolysis for beta-cell glucose signalingI D Dukes, M S McIntyre, R J Mertz, et al.The Journal of Biological Chemistry|November 11, 1994
Delayed rectifier K+ channel overexpression in transgenic islets and beta-cells associated with impaired glucose responsivenessL H Philipson, M P Rosenberg, A Kuznetsov, et al.The Journal of Allergy and Clinical Immunology|December 5, 2002
beta-Agonists and metabolismL H PhilipsonHormone and Metabolic Research = Hormon- Und Stoffwechselforschung = Hormones Et Metabolisme|September 24, 1999
Beta-cell ion channels: keys to endodermal excitabilityL H PhilipsonThe American Journal of Physiology|January 11, 1996
NIDDM is associated with loss of pancreatic beta-cell L-type Ca2+ channel activityM W Roe, J F Worley, Y Tokuyama, et al.The Journal of Biological Chemistry|July 15, 1994
Defective glucose-dependent endoplasmic reticulum Ca2+ sequestration in diabetic mouse islets of LangerhansM W Roe, L H Philipson, C J Frangakis, et al.Journal of Molecular and Cellular Cardiology|January 1, 1984
An analysis of the rate-dependent action of lidoflazine in mammalian sino-atrial node and Purkinje fibresG Hart, I D DukesThe American Journal of Physiology|November 1, 1989
Tedisamil inactivates transient outward K+ current in rat ventricular myocytesI D Dukes, M MoradThe Journal of Physiology|April 1, 1991
The transient K+ current in rat ventricular myocytes: evaluation of its Ca2+ and Na+ dependenceI D Dukes, M MoradPageof 7