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I Olazabal

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Molecular Endocrinology (Baltimore, Md.)|April 19, 2000
Prolactin (PRL)-PRL receptor system increases cell proliferation involving JNK (c-Jun amino terminal kinase) and AP-1 activation: inhibition by glucocorticoidsI Olazabal, J Muñoz, S Ogueta, et al.
The Journal of Endocrinology|May 16, 2000
Transgenic mice expressing bovine GH develop arthritic disorder and self-antibodiesS Ogueta, I Olazabal, I Santos, et al.
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Showing results (1-10 of 2) with videos related to

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Pageof 1
Molecular Endocrinology (Baltimore, Md.)|April 19, 2000
Prolactin (PRL)-PRL receptor system increases cell proliferation involving JNK (c-Jun amino terminal kinase) and AP-1 activation: inhibition by glucocorticoidsI Olazabal, J Muñoz, S Ogueta, et al.
The Journal of Endocrinology|May 16, 2000
Transgenic mice expressing bovine GH develop arthritic disorder and self-antibodiesS Ogueta, I Olazabal, I Santos, et al.
Pageof 1