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Ignacio V Sandoval

Showing results (1-10 of 15) with videos related to

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Journal of Cellular Physiology|March 28, 2009
The Cdk5 inhibitor roscovitine strongly inhibits glucose uptake in 3T3-L1 adipocytes without altering GLUT4 translocation from internal pools to the cell surfaceGemma Muruáis, Vassiliki Lalioti, Ignacio V Sandoval
Seminars in Liver Disease|June 1, 2017
Disorders in Hepatic Copper Secretion: Wilson's Disease and Pleomorphic SyndromesVasiliki Lalioti, Akihito Tsubota, Ignacio V Sandoval
Cell Cycle (Georgetown, Tex.)|January 12, 2010
Cdk5, the multifunctional surveyorVassiliki Lalioti, Diego Pulido, Ignacio V Sandoval
Traffic (Copenhagen, Denmark)|May 17, 2014
DKWSLLL, a versatile DXXXLL-type signal with distinct roles in the Cu(+)-regulated trafficking of ATP7BVasiliki Lalioti, Sonia Hernandez-Tiedra, Ignacio V Sandoval
The Journal of Biological Chemistry|February 14, 2002
The insulin-sensitive glucose transporter, GLUT4, interacts physically with Daxx. Two proteins with capacity to bind Ubc9 and conjugated to SUMO1Vassiliki S Lalioti, Silvia Vergarajauregui, Diego Pulido, et al.
Traffic (Copenhagen, Denmark)|October 25, 2021
The E-Syt3 cleavage and traffic uncovers the primordial cisterna, a new organelle that mothers the lipid droplets in the adipocyteVasiliki Lalioti, Galina V Beznoussenko, Alexander A Mironov, et al.
Frontiers in Bioscience (Landmark Edition)|June 2, 2009
Molecular mechanisms of copper homeostasisVasiliki Lalioti, Gemma Muruais, Yo Tsuchiya, et al.
Journal of Cellular Physiology|March 6, 2013
C6orf89 encodes three distinct HDAC enhancers that function in the nucleolus, the golgi and the midbodyVasiliki S Lalioti, Silvia Vergarajauregui, Alfredo Villasante, et al.
Journal of Cellular Physiology|October 22, 2008
Daxx functions as a scaffold of a protein assembly constituted by GLUT4, JNK1 and KIF5BVasiliki S Lalioti, Silvia Vergarajauregui, Yo Tsuchiya, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 4, 2009
The atypical kinase Cdk5 is activated by insulin, regulates the association between GLUT4 and E-Syt1, and modulates glucose transport in 3T3-L1 adipocytesVasiliki Lalioti, Gemma Muruais, Ana Dinarina, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Journal of Cellular Physiology|March 28, 2009
The Cdk5 inhibitor roscovitine strongly inhibits glucose uptake in 3T3-L1 adipocytes without altering GLUT4 translocation from internal pools to the cell surfaceGemma Muruáis, Vassiliki Lalioti, Ignacio V Sandoval
Seminars in Liver Disease|June 1, 2017
Disorders in Hepatic Copper Secretion: Wilson's Disease and Pleomorphic SyndromesVasiliki Lalioti, Akihito Tsubota, Ignacio V Sandoval
Cell Cycle (Georgetown, Tex.)|January 12, 2010
Cdk5, the multifunctional surveyorVassiliki Lalioti, Diego Pulido, Ignacio V Sandoval
Traffic (Copenhagen, Denmark)|May 17, 2014
DKWSLLL, a versatile DXXXLL-type signal with distinct roles in the Cu(+)-regulated trafficking of ATP7BVasiliki Lalioti, Sonia Hernandez-Tiedra, Ignacio V Sandoval
The Journal of Biological Chemistry|February 14, 2002
The insulin-sensitive glucose transporter, GLUT4, interacts physically with Daxx. Two proteins with capacity to bind Ubc9 and conjugated to SUMO1Vassiliki S Lalioti, Silvia Vergarajauregui, Diego Pulido, et al.
Traffic (Copenhagen, Denmark)|October 25, 2021
The E-Syt3 cleavage and traffic uncovers the primordial cisterna, a new organelle that mothers the lipid droplets in the adipocyteVasiliki Lalioti, Galina V Beznoussenko, Alexander A Mironov, et al.
Frontiers in Bioscience (Landmark Edition)|June 2, 2009
Molecular mechanisms of copper homeostasisVasiliki Lalioti, Gemma Muruais, Yo Tsuchiya, et al.
Journal of Cellular Physiology|March 6, 2013
C6orf89 encodes three distinct HDAC enhancers that function in the nucleolus, the golgi and the midbodyVasiliki S Lalioti, Silvia Vergarajauregui, Alfredo Villasante, et al.
Journal of Cellular Physiology|October 22, 2008
Daxx functions as a scaffold of a protein assembly constituted by GLUT4, JNK1 and KIF5BVasiliki S Lalioti, Silvia Vergarajauregui, Yo Tsuchiya, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 4, 2009
The atypical kinase Cdk5 is activated by insulin, regulates the association between GLUT4 and E-Syt1, and modulates glucose transport in 3T3-L1 adipocytesVasiliki Lalioti, Gemma Muruais, Ana Dinarina, et al.
Pageof 2