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J Barritt

Showing results (51-60 of 127) with videos related to

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The Biochemical Journal|September 6, 2001
A diacylglycerol-activated Ca2+ channel in PC12 cells (an adrenal chromaffin cell line) correlates with expression of the TRP-6 (transient receptor potential) proteinY Tesfai, H M Brereton, G J Barritt
The Biochemical Journal|September 1, 1975
Stimulation of hepatic mitochondrial calcium transport by elevated plasma insulin concentrationsD M Dorman, G J Barritt, F L Bygrave
The Biochemical Journal|January 15, 1983
Evidence that glucagon acts on the liver to decrease mitochondrial calcium storesH M Baddams, L B Chang, G J Barritt
The Biochemical Journal|May 23, 1998
Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondriaK C Fernando, R B Gregory, G J Barritt
Cell Calcium|August 12, 2019
Deranged hepatocyte intracellular Ca<sup>2+</sup> homeostasis and the progression of non-alcoholic fatty liver disease to hepatocellular carcinomaEunus S Ali, Grigori Y Rychkov, Greg J Barritt
The Biochemical Journal|October 15, 1983
A kinetic investigation of the effects of adrenaline on 45Ca2+ exchange in isolated hepatocytes at different Ca2+ concentrations, at 20 degrees C and in the presence of inhibitors of mitochondrial Ca2+ transportJ C Parker, G J Barritt, J C Wadsworth
The Biochemical Journal|June 15, 1978
Effects of hormones and N6O2'-dibutyryl-adenosine 3' :5'-cyclic monophosphate, administered in vivo, on phosphate transport and metabolism in isolated rat liver mitochondriaG J Barritt, R F Thorne, B P Hughes
The Journal of Biological Chemistry|April 29, 2000
Regulation of F-actin and endoplasmic reticulum organization by the trimeric G-protein Gi2 in rat hepatocytes. Implication for the activation of store-operated Ca2+ inflowY J Wang, R B Gregory, G J Barritt
The Biochemical Journal|September 15, 1991
Inhibition of Ca2+ inflow causes an abrupt cessation of growth-factor-induced repetitive free Ca2+ transients in single NIH-3T3 cellsA J Polverino, B P Hughes, G J Barritt
Cancers|September 29, 2020
Targeting Ca<sup>2+</sup> Signaling in the Initiation, Promotion and Progression of Hepatocellular CarcinomaEunus S Ali, Grigori Y Rychkov, Greg J Barritt
Pageof 13

Showing results (51-60 of 127) with videos related to

Sort By:
Pageof 13
The Biochemical Journal|September 6, 2001
A diacylglycerol-activated Ca2+ channel in PC12 cells (an adrenal chromaffin cell line) correlates with expression of the TRP-6 (transient receptor potential) proteinY Tesfai, H M Brereton, G J Barritt
The Biochemical Journal|September 1, 1975
Stimulation of hepatic mitochondrial calcium transport by elevated plasma insulin concentrationsD M Dorman, G J Barritt, F L Bygrave
The Biochemical Journal|January 15, 1983
Evidence that glucagon acts on the liver to decrease mitochondrial calcium storesH M Baddams, L B Chang, G J Barritt
The Biochemical Journal|May 23, 1998
Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondriaK C Fernando, R B Gregory, G J Barritt
Cell Calcium|August 12, 2019
Deranged hepatocyte intracellular Ca<sup>2+</sup> homeostasis and the progression of non-alcoholic fatty liver disease to hepatocellular carcinomaEunus S Ali, Grigori Y Rychkov, Greg J Barritt
The Biochemical Journal|October 15, 1983
A kinetic investigation of the effects of adrenaline on 45Ca2+ exchange in isolated hepatocytes at different Ca2+ concentrations, at 20 degrees C and in the presence of inhibitors of mitochondrial Ca2+ transportJ C Parker, G J Barritt, J C Wadsworth
The Biochemical Journal|June 15, 1978
Effects of hormones and N6O2'-dibutyryl-adenosine 3' :5'-cyclic monophosphate, administered in vivo, on phosphate transport and metabolism in isolated rat liver mitochondriaG J Barritt, R F Thorne, B P Hughes
The Journal of Biological Chemistry|April 29, 2000
Regulation of F-actin and endoplasmic reticulum organization by the trimeric G-protein Gi2 in rat hepatocytes. Implication for the activation of store-operated Ca2+ inflowY J Wang, R B Gregory, G J Barritt
The Biochemical Journal|September 15, 1991
Inhibition of Ca2+ inflow causes an abrupt cessation of growth-factor-induced repetitive free Ca2+ transients in single NIH-3T3 cellsA J Polverino, B P Hughes, G J Barritt
Cancers|September 29, 2020
Targeting Ca<sup>2+</sup> Signaling in the Initiation, Promotion and Progression of Hepatocellular CarcinomaEunus S Ali, Grigori Y Rychkov, Greg J Barritt
Pageof 13