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Oncogene
|
October 15, 2013
The MYB proto-oncogene suppresses monocytic differentiation of acute myeloid leukemia cells via transcriptional activation of its target gene GFI1
L Zhao, P Ye, T J Gonda
Scientific Reports
|
December 29, 2019
The mechanism of MYB transcriptional regulation by MLL-AF9 oncoprotein
Lu Cao, Partha Mitra, Thomas J Gonda
Leukemia Research
|
February 13, 2013
The MYB oncogene can suppress apoptosis in acute myeloid leukemia cells by transcriptional repression of DRAK2 expression
Ping Ye, Liang Zhao, Thomas J Gonda
The EMBO Journal
|
June 1, 1989
Activation of c-myb by carboxy-terminal truncation: relationship to transformation of murine haemopoietic cells in vitro
T J Gonda, C Buckmaster, R G Ramsay
The EMBO Journal
|
September 1, 1995
Activating point mutations in the common beta subunit of the human GM-CSF, IL-3 and IL-5 receptors suggest the involvement of beta subunit dimerization and cell type-specific molecules in signalling
B J Jenkins, R D'Andrea, T J Gonda
Oncogene
|
October 1, 1991
Increase in specific DNA binding by carboxyl truncation suggests a mechanism for activation of Myb
R G Ramsay, S Ishii, T J Gonda
Contemporary Nurse
|
January 6, 2001
Dedicated education units: 1. A new concept for clinical teaching and learning
K Edgecombe, K Wotton, J Gonda, et al.
Expert Opinion on Biological Therapy
|
May 15, 2008
Estrogen and MYB in breast cancer: potential for new therapies
Thomas J Gonda, Paul Leo, Robert G Ramsay
The Journal of Biological Chemistry
|
March 20, 1999
A cell type-specific constitutive point mutant of the common beta-subunit of the human granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukin (IL)-3, and IL-5 receptors requires the GM-CSF receptor alpha-subunit for activation
B J Jenkins, F Le, T J Gonda
Breast Cancer Research : BCR
|
July 28, 2010
MYB suppresses differentiation and apoptosis of human breast cancer cells
Yvette Drabsch, Ramsay G Robert, Thomas J Gonda
Page
of 14
Search research articles
Search
Showing results (21-30 of 139) with videos related to
Sort By:
Page
of 14
Oncogene
|
October 15, 2013
The MYB proto-oncogene suppresses monocytic differentiation of acute myeloid leukemia cells via transcriptional activation of its target gene GFI1
L Zhao, P Ye, T J Gonda
Scientific Reports
|
December 29, 2019
The mechanism of MYB transcriptional regulation by MLL-AF9 oncoprotein
Lu Cao, Partha Mitra, Thomas J Gonda
Leukemia Research
|
February 13, 2013
The MYB oncogene can suppress apoptosis in acute myeloid leukemia cells by transcriptional repression of DRAK2 expression
Ping Ye, Liang Zhao, Thomas J Gonda
The EMBO Journal
|
June 1, 1989
Activation of c-myb by carboxy-terminal truncation: relationship to transformation of murine haemopoietic cells in vitro
T J Gonda, C Buckmaster, R G Ramsay
The EMBO Journal
|
September 1, 1995
Activating point mutations in the common beta subunit of the human GM-CSF, IL-3 and IL-5 receptors suggest the involvement of beta subunit dimerization and cell type-specific molecules in signalling
B J Jenkins, R D'Andrea, T J Gonda
Oncogene
|
October 1, 1991
Increase in specific DNA binding by carboxyl truncation suggests a mechanism for activation of Myb
R G Ramsay, S Ishii, T J Gonda
Contemporary Nurse
|
January 6, 2001
Dedicated education units: 1. A new concept for clinical teaching and learning
K Edgecombe, K Wotton, J Gonda, et al.
Expert Opinion on Biological Therapy
|
May 15, 2008
Estrogen and MYB in breast cancer: potential for new therapies
Thomas J Gonda, Paul Leo, Robert G Ramsay
The Journal of Biological Chemistry
|
March 20, 1999
A cell type-specific constitutive point mutant of the common beta-subunit of the human granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukin (IL)-3, and IL-5 receptors requires the GM-CSF receptor alpha-subunit for activation
B J Jenkins, F Le, T J Gonda
Breast Cancer Research : BCR
|
July 28, 2010
MYB suppresses differentiation and apoptosis of human breast cancer cells
Yvette Drabsch, Ramsay G Robert, Thomas J Gonda
Page
of 14